Wilhelm D - Search Results

1

Activation of CD95 (APO-1/Fas) signaling by ceramide mediates cancer therapy-induced apoptosis.

Herr I, Wilhelm D, Böhler T, Angel P, Debatin KM. Herr I, et al. Among authors: wilhelm d. EMBO J. 1997 Oct 15;16(20):6200-8. doi: 10.1093/emboj/16.20.6200. EMBO J. 1997. PMID: 9321399 Free PMC article.

2

JNK/SAPK activity is not sufficient for anticancer therapy-induced apoptosis involving CD95-L, TRAIL and TNF-alpha.

Herr I, Wilhelm D, Böhler T, Angel P, Debatin KM. Herr I, et al. Among authors: wilhelm d. Int J Cancer. 1999 Jan 29;80(3):417-24. doi: 10.1002/(sici)1097-0215(19990129)80:3<417::aid-ijc14>3.0.co;2-b. Int J Cancer. 1999. PMID: 9935184

3

JNK/SAPK activity contributes to TRAIL-induced apoptosis.

Herr I, Wilhelm D, Meyer E, Jeremias I, Angel P, Debatin KM. Herr I, et al. Among authors: wilhelm d. Cell Death Differ. 1999 Feb;6(2):130-5. doi: 10.1038/sj.cdd.4400467. Cell Death Differ. 1999. PMID: 10200559

4

ATF-2 is preferentially activated by stress-activated protein kinases to mediate c-jun induction in response to genotoxic agents.

van Dam H, Wilhelm D, Herr I, Steffen A, Herrlich P, Angel P. van Dam H, et al. Among authors: wilhelm d. EMBO J. 1995 Apr 18;14(8):1798-811. doi: 10.1002/j.1460-2075.1995.tb07168.x. EMBO J. 1995. PMID: 7737130 Free PMC article.

5

The level of intracellular glutathione is a key regulator for the induction of stress-activated signal transduction pathways including Jun N-terminal protein kinases and p38 kinase by alkylating agents.

Wilhelm D, Bender K, Knebel A, Angel P. Wilhelm D, et al. Mol Cell Biol. 1997 Aug;17(8):4792-800. doi: 10.1128/MCB.17.8.4792. Mol Cell Biol. 1997. PMID: 9234735 Free PMC article.

6

NFkappaB activation is required for interferon regulatory factor-1-mediated interferon beta induction.

Kirchhoff S, Wilhelm D, Angel P, Hauser H. Kirchhoff S, et al. Among authors: wilhelm d. Eur J Biochem. 1999 Apr;261(2):546-54. doi: 10.1046/j.1432-1327.1999.00308.x. Eur J Biochem. 1999. PMID: 10215868 Free article.

7

Both ATF-2 and c-Jun are phosphorylated by stress-activated protein kinases in response to UV irradiation.

Wilhelm D, van Dam H, Herr I, Baumann B, Herrlich P, Angel P. Wilhelm D, et al. Immunobiology. 1995 Jul;193(2-4):143-8. doi: 10.1016/S0171-2985(11)80537-1. Immunobiology. 1995. PMID: 8530137 Review. No abstract available.

8

Parathyroid hormone inhibits c-Jun N-terminal kinase activity in rat osteoblastic cells by a protein kinase A-dependent pathway.

Doggett TA, Swarthout JT, Jefcoat SC Jr, Wilhelm D, Dieckmann A, Angel P, Partridge NC. Doggett TA, et al. Among authors: wilhelm d. Endocrinology. 2002 May;143(5):1880-8. doi: 10.1210/endo.143.5.8759. Endocrinology. 2002. PMID: 11956171

9

Stimulation of c-Jun activity by CBP: c-Jun residues Ser63/73 are required for CBP induced stimulation in vivo and CBP binding in vitro.

Bannister AJ, Oehler T, Wilhelm D, Angel P, Kouzarides T. Bannister AJ, et al. Among authors: wilhelm d. Oncogene. 1995 Dec 21;11(12):2509-14. Oncogene. 1995. PMID: 8545107

10

The Wilms' tumor gene Wt1 is required for normal development of the retina.

Wagner KD, Wagner N, Vidal VP, Schley G, Wilhelm D, Schedl A, Englert C, Scholz H. Wagner KD, et al. Among authors: wilhelm d. EMBO J. 2002 Mar 15;21(6):1398-405. doi: 10.1093/emboj/21.6.1398. EMBO J. 2002. PMID: 11889045 Free PMC article.

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