APC/CCDH1 synchronizes ribose-5-phosphate levels and DNA synthesis to cell cycle progression

Nat Commun. 2019 Jun 7;10(1):2502. doi: 10.1038/s41467-019-10375-x.

Abstract

Accumulation of nucleotide building blocks prior to and during S phase facilitates DNA duplication. Herein, we find that the anaphase-promoting complex/cyclosome (APC/C) synchronizes ribose-5-phosphate levels and DNA synthesis during the cell cycle. In late G1 and S phases, transketolase-like 1 (TKTL1) is overexpressed and forms stable TKTL1-transketolase heterodimers that accumulate ribose-5-phosphate. This accumulation occurs by asymmetric production of ribose-5-phosphate from the non-oxidative pentose phosphate pathway and prevention of ribose-5-phosphate removal by depleting transketolase homodimers. In the G2 and M phases after DNA synthesis, expression of the APC/C adaptor CDH1 allows APC/CCDH1 to degrade D-box-containing TKTL1, abrogating ribose-5-phosphate accumulation by TKTL1. TKTL1-overexpressing cancer cells exhibit elevated ribose-5-phosphate levels. The low CDH1 or high TKTL1-induced accumulation of ribose-5-phosphate facilitates nucleotide and DNA synthesis as well as cell cycle progression in a ribose-5-phosphate-saturable manner. Here we reveal that the cell cycle control machinery regulates DNA synthesis by mediating ribose-5-phosphate sufficiency.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Anaphase-Promoting Complex-Cyclosome / metabolism*
  • Cdc20 Proteins / metabolism
  • Cdh1 Proteins / metabolism*
  • Cell Cycle Proteins / metabolism
  • Cell Cycle*
  • Cell Division
  • DNA Replication*
  • G2 Phase
  • Humans
  • Pentose Phosphate Pathway
  • Ribosemonophosphates / metabolism*
  • S Phase
  • Transketolase / metabolism*

Substances

  • Cdc20 Proteins
  • Cdh1 Proteins
  • Cell Cycle Proteins
  • Ribosemonophosphates
  • ribose-5-phosphate
  • TKTL1 protein, human
  • Transketolase
  • Anaphase-Promoting Complex-Cyclosome