Insulin antibodies retard and insulin accelerates growth and differentiation in early embryos

Diabetes. 1985 Oct;34(10):1063-7. doi: 10.2337/diab.34.10.1063.

Abstract

The physiologic function of insulin in early embryonic life is unknown. We have shown that insulin is present in unfertilized eggs and in chick embryos at 2-3 days of development, even before the emergence of the endocrine pancreas. To define insulin's role, we exposed 2-day-old chick embryos to anti-insulin antibodies and followed their development up to day 5. Antibody-treated embryos had a higher rate of growth retardation and death by days 3-5 of embryogenesis, compared with controls. Among the survivors, biochemical maturation was delayed at days 4 and 5; weight, protein, total creatine kinase activity, and creatine kinase-MB were decreased in antibody-treated embryos. By contrast, insulin (50 ng/embryo) administered to 2-day-old embryos yielded nearly symmetrical stimulatory results. These findings suggest that endogenous insulin plays a probable physiologic role regulating growth and differentiation in early embryos. In addition, the findings provide some clues to a possible function for insulin produced outside the organism's own beta cells.

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Animals
  • Chick Embryo
  • Creatine Kinase / analysis
  • Embryonic and Fetal Development / drug effects*
  • Insulin / pharmacology*
  • Insulin Antibodies / pharmacology*
  • Proteins / analysis
  • Time Factors

Substances

  • Insulin
  • Insulin Antibodies
  • Proteins
  • Creatine Kinase