CA69161/CA/NCI NIH HHS/United States[Grants and Funding] - Search Results

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Subject: CA69161/CA/NCI NIH HHS/United States[Grants and F - PubMed

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Identification of a novel class of genomic DNA-binding sites suggests a mechanism for selectivity in target gene activation by the tumor suppressor protein p53.

Resnick-Silverman L, St Clair S, Maurer M, Zhao K, Manfredi JJ. Resnick-Silverman L, et al. Genes Dev. 1998 Jul 15;12(14):2102-7. doi: 10.1101/gad.12.14.2102. Genes Dev. 1998. PMID: 9679054 Free PMC article.

The tumor suppressor protein p53 requires a cofactor to activate transcriptionally the human BAX promoter.

Thornborrow EC, Manfredi JJ. Thornborrow EC, et al. J Biol Chem. 2001 May 11;276(19):15598-608. doi: 10.1074/jbc.M011643200. Epub 2001 Feb 13. J Biol Chem. 2001. PMID: 11278953 Free article.

One mechanism for cell type-specific regulation of the bax promoter by the tumor suppressor p53 is dictated by the p53 response element.

Thornborrow EC, Manfredi JJ. Thornborrow EC, et al. J Biol Chem. 1999 Nov 19;274(47):33747-56. doi: 10.1074/jbc.274.47.33747. J Biol Chem. 1999. PMID: 10559267 Free article.

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