R01 AR044719/AR/NIAMS NIH HHS/United States[Grants and Funding] - Search Results

Year	Number of Results
2002	1
2004	1
2006	4
2007	3
2008	2
2009	3
2012	1
2013	2
2014	1
2016	2
2018	4
2019	1
2024	0

1

Genetics and genomics of osteoclast differentiation: integrating cell signaling pathways and gene networks.

Sharma SM, Hu R, Bronisz A, Meadows N, Lusby T, Fletcher B, Hume DA, Cassady AI, Ostrowski MC. Sharma SM, et al. Crit Rev Eukaryot Gene Expr. 2006;16(3):253-77. doi: 10.1615/critreveukargeneexpr.v16.i3.40. Crit Rev Eukaryot Gene Expr. 2006. PMID: 17073554 Review.

2

Eomes partners with PU.1 and MITF to Regulate Transcription Factors Critical for osteoclast differentiation.

Carey HA, Hildreth BE 3rd, Samuvel DJ, Thies KA, Rosol TJ, Toribio RE, Charles JF, Ostrowski MC, Sharma SM. Carey HA, et al. iScience. 2019 Jan 25;11:238-245. doi: 10.1016/j.isci.2018.12.018. Epub 2018 Dec 27. iScience. 2019. PMID: 30634169 Free PMC article.

3

Eos, MITF, and PU.1 recruit corepressors to osteoclast-specific genes in committed myeloid progenitors.

Hu R, Sharma SM, Bronisz A, Srinivasan R, Sankar U, Ostrowski MC. Hu R, et al. Mol Cell Biol. 2007 Jun;27(11):4018-27. doi: 10.1128/MCB.01839-06. Epub 2007 Apr 2. Mol Cell Biol. 2007. PMID: 17403896 Free PMC article.

4

MITF and PU.1 recruit p38 MAPK and NFATc1 to target genes during osteoclast differentiation.

Sharma SM, Bronisz A, Hu R, Patel K, Mansky KC, Sif S, Ostrowski MC. Sharma SM, et al. J Biol Chem. 2007 May 25;282(21):15921-9. doi: 10.1074/jbc.M609723200. Epub 2007 Apr 2. J Biol Chem. 2007. PMID: 17403683 Free article.

5

Enhancer variants reveal a conserved transcription factor network governed by PU.1 during osteoclast differentiation.

Carey HA, Hildreth BE 3rd, Geisler JA, Nickel MC, Cabrera J, Ghosh S, Jiang Y, Yan J, Lee J, Makam S, Young NA, Valiente GR, Jarjour WN, Huang K, Rosol TJ, Toribio RE, Charles JF, Ostrowski MC, Sharma SM. Carey HA, et al. Bone Res. 2018 Mar 28;6:8. doi: 10.1038/s41413-018-0011-1. eCollection 2018. Bone Res. 2018. PMID: 29619268 Free PMC article.

6

The multifunctional protein fused in sarcoma (FUS) is a coactivator of microphthalmia-associated transcription factor (MITF).

Bronisz A, Carey HA, Godlewski J, Sif S, Ostrowski MC, Sharma SM. Bronisz A, et al. J Biol Chem. 2014 Jan 3;289(1):326-34. doi: 10.1074/jbc.M113.493874. Epub 2013 Nov 20. J Biol Chem. 2014. PMID: 24257758 Free PMC article.

7

Microphthalmia-associated transcription factor interactions with 14-3-3 modulate differentiation of committed myeloid precursors.

Bronisz A, Sharma SM, Hu R, Godlewski J, Tzivion G, Mansky KC, Ostrowski MC. Bronisz A, et al. Mol Biol Cell. 2006 Sep;17(9):3897-906. doi: 10.1091/mbc.e06-05-0470. Epub 2006 Jul 5. Mol Biol Cell. 2006. PMID: 16822840 Free PMC article.

8

The expression of Clcn7 and Ostm1 in osteoclasts is coregulated by microphthalmia transcription factor.

Meadows NA, Sharma SM, Faulkner GJ, Ostrowski MC, Hume DA, Cassady AI. Meadows NA, et al. J Biol Chem. 2007 Jan 19;282(3):1891-904. doi: 10.1074/jbc.M608572200. Epub 2006 Nov 14. J Biol Chem. 2007. PMID: 17105730 Free article.

9

Stromal ETS2 Regulates Chemokine Production and Immune Cell Recruitment during Acinar-to-Ductal Metaplasia.

Pitarresi JR, Liu X, Sharma SM, Cuitiño MC, Kladney RD, Mace TA, Donohue S, Nayak SG, Qu C, Lee J, Woelke SA, Trela S, LaPak K, Yu L, McElroy J, Rosol TJ, Shakya R, Ludwig T, Lesinski GB, Fernandez SA, Konieczny SF, Leone G, Wu J, Ostrowski MC. Pitarresi JR, et al. Neoplasia. 2016 Sep;18(9):541-52. doi: 10.1016/j.neo.2016.07.006. Neoplasia. 2016. PMID: 27659014 Free PMC article.

10

NF-κB inhibition rescues cardiac function by remodeling calcium genes in a Duchenne muscular dystrophy model.

Peterson JM, Wang DJ, Shettigar V, Roof SR, Canan BD, Bakkar N, Shintaku J, Gu JM, Little SC, Ratnam NM, Londhe P, Lu L, Gaw CE, Petrosino JM, Liyanarachchi S, Wang H, Janssen PML, Davis JP, Ziolo MT, Sharma SM, Guttridge DC. Peterson JM, et al. Nat Commun. 2018 Aug 24;9(1):3431. doi: 10.1038/s41467-018-05910-1. Nat Commun. 2018. PMID: 30143619 Free PMC article.

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