Viral cardiomyopathy: Difference between revisions
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== COVID-19 Cardiomyopathy == |
== COVID-19 Cardiomyopathy == |
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Patients with [[COVID |
Patients with [[COVID-19]] frequently experience [[heart]] issues.<ref name=":0">{{Cite journal |last1=Razaghi |first1=Ali |last2=Szakos |first2=Attila |last3=Al-Shakarji |first3=Riham |last4=Björnstedt |first4=Mikael |last5=Szekely |first5=Laszlo |date=2022-12-31 |title=Morphological changes without histological myocarditis in hearts of COVID-19 deceased patients |journal=Scandinavian Cardiovascular Journal |volume=56 |issue=1 |pages=166–173 |doi=10.1080/14017431.2022.2085320 |issn=1401-7431 |pmid=35678649|s2cid=249521076 |doi-access=free }}</ref> According to studies, people who have had previous cardiovascular conditions like [[cardiomyopathy]], [[hypertension]], [[Coronary Heart Disease|coronary heart disease]], or [[arrhythmia]] are more likely to become critically ill from [[SARS-CoV-2]] infection. [[Myocarditis]] may result from a direct viral infection of the myocardium.<ref name=":0" /> Cardiovascular biomarkers like [[troponin]], [[lactate dehydrogenase]], high sensitivity amino-terminal [[B-type natriuretic peptide]], [[Creatine kinase|creatinine kinase]], and creatinine kinase myocardial band, which indicate myocardial damage, increase in concentration in response to COVID-19.<ref name=":0" /> Hundreds of studies have reported myocarditis/myopericarditis caused by COVID-19 infection in living patients, with a [[male]] predominance (58%), and a [[median age]] of 50 years. <ref name=":0" /> |
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== See also == |
== See also == |
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Latest revision as of 03:34, 29 May 2024
| Viral cardiomyopathy | |
|---|---|
| Specialty | Cardiology |
| Symptoms |
|
| Causes | viral infections cause myocarditis |
| Diagnostic method | Electrocardiography, Echocardiography, MRI[1] |
Viral cardiomyopathy occurs when viral infections cause myocarditis with a resulting thickening of the myocardium and dilation of the ventricles. These viruses include Coxsackie B and adenovirus, echoviruses, influenza H1N1, Epstein–Barr virus, rubella (German measles virus), varicella (chickenpox virus), mumps, measles, parvoviruses, yellow fever, dengue fever, polio, rabies and the viruses that cause hepatitis A and C,[2][3][4] as well as COVID-19,[5][6] which has been seen to cause this in persons otherwise thought to have a "low risk" of the virus's effects.[7]
COVID-19 Cardiomyopathy
[edit]Patients with COVID-19 frequently experience heart issues.[8] According to studies, people who have had previous cardiovascular conditions like cardiomyopathy, hypertension, coronary heart disease, or arrhythmia are more likely to become critically ill from SARS-CoV-2 infection. Myocarditis may result from a direct viral infection of the myocardium.[8] Cardiovascular biomarkers like troponin, lactate dehydrogenase, high sensitivity amino-terminal B-type natriuretic peptide, creatinine kinase, and creatinine kinase myocardial band, which indicate myocardial damage, increase in concentration in response to COVID-19.[8] Hundreds of studies have reported myocarditis/myopericarditis caused by COVID-19 infection in living patients, with a male predominance (58%), and a median age of 50 years. [8]
See also
[edit]References
[edit]- ^ Schultz, Jason C.; Hilliard, Anthony A.; Cooper, Leslie T.; Rihal, Charanjit S. (1985-01-01). "Diagnosis and Treatment of Viral Myocarditis". Mayo Clinic Proceedings. 84 (11): 1001–1009. doi:10.1016/S0025-6196(11)60670-8. PMC 2770911. PMID 19880690.
- ^ Barbandi M, Cordero-Reyes A, Orrego CM, Torre-Amione G, Seethamraju H (Jan 2012). "A case series of reversible acute cardiomyopathy associated with H1N1 influenza infection". Methodist DeBakey Cardiovascular Journal. 8 (1): 42–5. doi:10.14797/mdcj-8-1-42. PMC 3405785. PMID 22891110.
- ^ Badorff C; Lee G. H.; Knowlton K. U. (2000). "Enteroviral cardiomyopathy: bad news for the dystrophin-glycoprotein complex". Herz. 25 (3): 227–32. doi:10.1007/s000590050011. PMID 10904843. S2CID 25973717.
- ^ Mutlu H, Alam M, Ozbilgin OF (2011). "A rare case of Epstein-Barr virus-induced dilated cardiomyopathy". Heart & Lung. 40 (1): 81–7. doi:10.1016/j.hrtlng.2009.12.012. PMID 20561866.
- ^ Rathore, Sawai Singh; Rojas, Gianpier Alonzo; Sondhi, Manush; Pothuru, Suveenkrishna; Pydi, Reshma; Kancherla, Neeraj; Singh, Romil; Ahmed, Noman Khurshid; Shah, Jill; Tousif, Sohaib; Baloch, Unaiza Tariq (2021). "Myocarditis associated with Covid-19 disease: A systematic review of published case reports and case series". International Journal of Clinical Practice. 75 (11): e14470. doi:10.1111/ijcp.14470. ISSN 1742-1241. PMID 34235815. S2CID 235768792.
- ^ Siripanthong, Bhurint; Nazarian, Saman; Muser, Daniele; Deo, Rajat; Santangeli, Pasquale; Khanji, Mohammed Y.; Cooper, Leslie T.; Chahal, C. Anwar A. (September 2020). "Recognizing COVID-19–related myocarditis: The possible pathophysiology and proposed guideline for diagnosis and management". Heart Rhythm. 17 (9): 1463–1471. doi:10.1016/j.hrthm.2020.05.001. ISSN 1547-5271. PMC 7199677. PMID 32387246.
- ^ Siripanthong B, Nazarian S, Muser D, Deo R, Santangeli P, Khanji M, Cooper L, Jr, Chahal A (5 May 2020). "Recognizing COVID-19–related myocarditis: The possible pathophysiology and proposed guideline for diagnosis and management". Heart Rhythm. 17 (9): 1463–1471. doi:10.1016/j.hrthm.2020.05.001. PMC 7199677. PMID 32387246.
- ^ a b c d Razaghi, Ali; Szakos, Attila; Al-Shakarji, Riham; Björnstedt, Mikael; Szekely, Laszlo (2022-12-31). "Morphological changes without histological myocarditis in hearts of COVID-19 deceased patients". Scandinavian Cardiovascular Journal. 56 (1): 166–173. doi:10.1080/14017431.2022.2085320. ISSN 1401-7431. PMID 35678649. S2CID 249521076.