Activating transcription factor-2 is a positive regulator in CaM kinase IV-induced human insulin gene expression

Diabetes. 2000 Jul;49(7):1142-8. doi: 10.2337/diabetes.49.7.1142.

Abstract

Insulin plays a crucial role in the regulation of glucose-homeostasis, and its synthesis is regulated by several stimuli. The transcription of the human insulin gene, enhanced by an elevated intracellular concentration of calcium ions, was completely blocked by Ca2+/calmodulin-dependent protein kinase inhibitor. The activity of the transcription factor activating transcription factor-2 (ATF-2), which binds to the cAMP responsive elements of the human insulin gene, was enhanced by Ca2+/calmodulin-dependent protein kinase IV (CaMKIV). Mutagenesis studies showed that Thr69, Thr71, and Thr73 of ATF-2 are all required for activation by CaMKIV. CaMKIV-induced ATF-2 transcriptional activity was not altered by activation of cJun NH2-terminal protein kinase (JNK) or p38 mitogen-activated protein (MAP) kinase. Furthermore, when transfected into rat primary cultured islets, ATF-2 enhanced glucose-induced insulin promoter activity, whereas cAMP response element-binding protein (CREB) repressed it. These results suggest a mechanism in which ATF-2 regulates insulin gene expression in pancreatic beta-cells, with the transcriptional activity of ATF-2 being increased by an elevated concentration of calcium ions.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Activating Transcription Factor 2
  • Amino Acid Substitution
  • Animals
  • Calcium-Calmodulin-Dependent Protein Kinase Type 4
  • Calcium-Calmodulin-Dependent Protein Kinases / metabolism*
  • Cell Line
  • Cricetinae
  • Cyclic AMP Response Element-Binding Protein / chemistry
  • Cyclic AMP Response Element-Binding Protein / genetics
  • Cyclic AMP Response Element-Binding Protein / metabolism*
  • Gene Expression Regulation* / drug effects
  • Glucose / pharmacology
  • Humans
  • Insulin / genetics*
  • Islets of Langerhans / metabolism*
  • Luciferases / genetics
  • Male
  • Mice
  • Mutagenesis, Site-Directed
  • Promoter Regions, Genetic / drug effects
  • Rats
  • Rats, Wistar
  • Recombinant Proteins / chemistry
  • Recombinant Proteins / metabolism
  • Transcription Factors / chemistry
  • Transcription Factors / genetics
  • Transcription Factors / metabolism*
  • Transcriptional Activation
  • Transfection

Substances

  • ATF2 protein, human
  • Activating Transcription Factor 2
  • Atf2 protein, mouse
  • Atf2 protein, rat
  • Cyclic AMP Response Element-Binding Protein
  • Insulin
  • Recombinant Proteins
  • Transcription Factors
  • Luciferases
  • CAMK4 protein, human
  • Calcium-Calmodulin-Dependent Protein Kinase Type 4
  • Calcium-Calmodulin-Dependent Protein Kinases
  • Camk4 protein, mouse
  • Camk4 protein, rat
  • Glucose