Apolipoprotein A4-1/2 polymorphism and response of serum lipids to dietary cholesterol in humans

J Lipid Res. 2000 Oct;41(10):1623-8.

Abstract

The response of serum lipids to dietary changes is to some extent an innate characteristic. One candidate genetic factor that may affect the response of serum lipids to a change in cholesterol intake is variation in the apolipoprotein A4 gene, known as the APOA4-1/2 or apoA-IVGln360His polymorphism. However, previous studies showed inconsistent results. We therefore fed 10 men and 23 women with the APOA4-1/1 genotype and 4 men and 13 women with the APOA4-1/2 or -2/2 genotype (carriers of the APOA4-2 allele) two diets high in saturated fat, one containing cholesterol at 12.4 mg/MJ, 136.4 mg/day, and one containing cholesterol at 86.2 mg/MJ, 948.2 mg/day. Each diet was supplied for 29 days in crossover design. The mean response of serum low density lipoprotein cholesterol was 0.44 mmol/l (17 mg/dl) in both subjects with the APOA4-1/1 genotype and in subjects with the APOA4-2 allele [95% confidence interval of difference in response, -0.20 to 0.19 mmol/l (-8 to 7 mg/dl)]. The mean response of high density lipoprotein cholesterol was also similar, 0.10 mmol/l (4 mg/dl), in the two APOA-4 genotype groups [95% confidence interval of difference in response, -0.07 to 0.08 mmol/l (-3 to 3 mg/dl)]. Thus, the APOA4-1/2 polymorphism did not affect the response of serum lipids to a change in the intake of cholesterol in this group of healthy Dutch subjects who consumed a background diet high in saturated fat. Knowledge of the APOA4-1/2 polymorphism is probably not a generally applicable tool for the identification of subjects who respond to a change in cholesterol intake.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adolescent
  • Adult
  • Antioxidants / pharmacology
  • Apolipoproteins A / blood
  • Apolipoproteins A / genetics*
  • Apolipoproteins A / pharmacology
  • Child
  • Cholesterol / blood
  • Cholesterol, Dietary / pharmacology*
  • Cholesterol, HDL / blood
  • Cholesterol, HDL / drug effects
  • Cholesterol, LDL / blood
  • Cholesterol, LDL / drug effects
  • Cross-Over Studies
  • Female
  • Genetic Variation
  • Genotype
  • Humans
  • Lipids / blood*
  • Male
  • Polymorphism, Genetic*
  • Protein Isoforms / blood
  • Protein Isoforms / genetics
  • Protein Isoforms / pharmacology

Substances

  • Antioxidants
  • Apolipoproteins A
  • Cholesterol, Dietary
  • Cholesterol, HDL
  • Cholesterol, LDL
  • Lipids
  • Protein Isoforms
  • apolipoprotein A-IV
  • Cholesterol