[Effects of homocysteine on human vascular endothelial cells, platelet aggregation and heparin cofactor activity]

Zhonghua Xue Ye Xue Za Zhi. 1999 Sep;20(9):471-3.
[Article in Chinese]

Abstract

Objectives: To elucidate partly the mechanism of homocysteine-associated thrombosis.

Methods: 1. Thrombomodulin (TM) antigen distribution on human vascular endothelial cell (EC) membrane was assayed by immune-fluorescence histochemistry stain and flow cytometry; 2. Plasma heparin cofactor activity was measured by chromogenic method.

Results: Homocysteine (Hcy) significantly down-regulated the expression of TM on EC, with a concentration-dependent manner; 2. Hcy did not influence platelet aggregation induced by ADP and thrombin; 3. Hcy exerted no effects on the activities of antithrombin III or heparin cofactor.

Conclusion: The effect of Hcy in developing thrombosis may be associated with an inhibitory action on EC TM expression; which was independent of platelet aggregation or heparin cofactor activity.

Publication types

  • English Abstract
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adult
  • Antithrombin III / metabolism
  • Cells, Cultured
  • Endothelium, Vascular / cytology*
  • Female
  • Heparin Cofactor II / metabolism*
  • Homocysteine / pharmacology*
  • Humans
  • Male
  • Platelet Aggregation / drug effects*
  • Random Allocation
  • Thrombomodulin / biosynthesis*
  • Umbilical Veins / cytology

Substances

  • Thrombomodulin
  • Homocysteine
  • Heparin Cofactor II
  • Antithrombin III