Tumstatin, an endothelial cell-specific inhibitor of protein synthesis

Science. 2002 Jan 4;295(5552):140-3. doi: 10.1126/science.1065298.

Abstract

Tumstatin is a 28-kilodalton fragment of type IV collagen that displays both anti-angiogenic and proapoptotic activity. Here we show that tumstatin functions as an endothelial cell-specific inhibitor of protein synthesis. Through a requisite interaction with alphaVbeta3 integrin, tumstatin inhibits activation of focal adhesion kinase (FAK), phosphatidylinositol 3-kinase (PI3-kinase), protein kinase B (PKB/Akt), and mammalian target of rapamycin (mTOR), and it prevents the dissociation of eukaryotic initiation factor 4E protein (eIF4E) from 4E-binding protein 1. These results establish a role for integrins in mediating cell-specific inhibition of cap-dependent protein synthesis and suggest a potential mechanism for tumstatin's selective effects on endothelial cells.

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Adaptor Proteins, Signal Transducing
  • Amino Acid Sequence
  • Animals
  • Autoantigens / chemistry
  • Autoantigens / metabolism
  • Autoantigens / pharmacology*
  • Carrier Proteins / metabolism
  • Cattle
  • Cell Cycle Proteins
  • Cells, Cultured
  • Collagen Type IV / chemistry
  • Collagen Type IV / metabolism
  • Collagen Type IV / pharmacology*
  • Endothelium, Vascular / cytology*
  • Endothelium, Vascular / drug effects
  • Endothelium, Vascular / metabolism*
  • Enzyme Activation / drug effects
  • Eukaryotic Initiation Factor-4E
  • Eukaryotic Initiation Factors
  • Focal Adhesion Kinase 1
  • Focal Adhesion Protein-Tyrosine Kinases
  • Humans
  • Mice
  • Molecular Sequence Data
  • Peptide Fragments / pharmacology
  • Peptide Initiation Factors / metabolism
  • Phosphatidylinositol 3-Kinases / metabolism
  • Phosphoproteins / metabolism
  • Phosphorylation
  • Protein Biosynthesis* / drug effects
  • Protein Kinase Inhibitors
  • Protein Kinases / metabolism
  • Protein Serine-Threonine Kinases*
  • Protein Synthesis Inhibitors / pharmacology*
  • Protein-Tyrosine Kinases / metabolism
  • Proto-Oncogene Proteins / metabolism
  • Proto-Oncogene Proteins c-akt
  • RNA Caps / metabolism
  • RNA, Messenger / genetics
  • RNA, Messenger / metabolism
  • Receptors, Vitronectin / metabolism
  • Signal Transduction
  • TOR Serine-Threonine Kinases
  • Tumor Cells, Cultured

Substances

  • Adaptor Proteins, Signal Transducing
  • Autoantigens
  • Carrier Proteins
  • Cell Cycle Proteins
  • Collagen Type IV
  • EIF4EBP1 protein, human
  • Eif4ebp1 protein, mouse
  • Eukaryotic Initiation Factor-4E
  • Eukaryotic Initiation Factors
  • Peptide Fragments
  • Peptide Initiation Factors
  • Phosphoproteins
  • Protein Kinase Inhibitors
  • Protein Synthesis Inhibitors
  • Proto-Oncogene Proteins
  • RNA Caps
  • RNA, Messenger
  • Receptors, Vitronectin
  • type IV collagen alpha3 chain
  • Protein Kinases
  • MTOR protein, human
  • mTOR protein, mouse
  • Protein-Tyrosine Kinases
  • Focal Adhesion Kinase 1
  • Focal Adhesion Protein-Tyrosine Kinases
  • PTK2 protein, human
  • Ptk2 protein, mouse
  • AKT1 protein, human
  • Protein Serine-Threonine Kinases
  • Proto-Oncogene Proteins c-akt
  • TOR Serine-Threonine Kinases