Synergistic engagement of an ineffective endogenous anti-tumor immune response and induction of IFN-gamma and Fas-ligand-dependent tumor eradication by combined administration of IL-18 and IL-2

J Immunol. 2002 Oct 15;169(8):4467-74. doi: 10.4049/jimmunol.169.8.4467.

Abstract

IFN-gamma is a critical component of the endogenous and many cytokine-induced antitumor immune responses. In this study we have shown that the combination of IL-18 and IL-2 (IL-18/IL-2) synergistically enhances IFN-gamma production both in vitro and in vivo, and synergizes in vivo to induce complete durable regression of well-established 3LL tumors in >80% of treated mice. We have observed a nascent, but ineffective, host immune response against 3LL that depends on endogenous IFN-gamma and IL-12 production and the Fas/Fas ligand (Fas-L) pathway. The combined administration of IL-18/IL-2 engages this endogenous response to induce tumor regression via a mechanism that is independent of NK and NKT cells or IL-12, but is critically dependent on CD8(+) T cells, IFN-gamma, and the Fas/Fas-L pathway. These studies demonstrate the importance of IFN-gamma as well as the Fas/Fas-L pathway in both endogenous and cytokine-driven antitumor immune responses engaged by IL-18/IL-2 and provide preclinical impetus for clinical investigation of this potent anti-tumor combination.

Publication types

  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Adjuvants, Immunologic / administration & dosage
  • Adjuvants, Immunologic / pharmacology
  • Animals
  • Antineoplastic Combined Chemotherapy Protocols / therapeutic use*
  • Apoptosis / immunology*
  • B-Lymphocytes / immunology
  • Carcinoma, Lewis Lung / immunology*
  • Carcinoma, Lewis Lung / pathology
  • Carcinoma, Lewis Lung / prevention & control*
  • Cells, Cultured
  • Drug Synergism
  • Fas Ligand Protein
  • Injections, Intraperitoneal
  • Interferon-gamma / biosynthesis*
  • Interferon-gamma / physiology
  • Interleukin-12 / physiology
  • Interleukin-18 / administration & dosage
  • Interleukin-18 / pharmacology
  • Interleukin-2 / administration & dosage
  • Interleukin-2 / pharmacology
  • Killer Cells, Natural / immunology
  • Ligands
  • Membrane Glycoproteins / physiology*
  • Mice
  • Mice, Inbred C57BL
  • Mice, Knockout
  • Mice, SCID
  • Remission Induction
  • Signal Transduction / immunology
  • Spleen / cytology
  • Spleen / immunology
  • Spleen / metabolism
  • T-Lymphocyte Subsets / immunology
  • fas Receptor / metabolism*
  • fas Receptor / physiology

Substances

  • Adjuvants, Immunologic
  • Fas Ligand Protein
  • Fasl protein, mouse
  • Interleukin-18
  • Interleukin-2
  • Ligands
  • Membrane Glycoproteins
  • fas Receptor
  • Interleukin-12
  • Interferon-gamma