Cytotoxic necrotizing factor 1 enhances reactive oxygen species-dependent transcription and secretion of proinflammatory cytokines in human uroepithelial cells

Infect Immun. 2003 Jul;71(7):4178-81. doi: 10.1128/IAI.71.7.4178-4181.2003.

Abstract

Uropathogenic Escherichia coli strains frequently produce a Rho-activating protein toxin named cytotoxic necrotizing factor type 1 (CNF1). We herein report that CNF1 promotes transcription and release of tumor necrosis factor alpha, gamma interferon, interleukin-6 (IL-6), and IL-8 proinflammatory cytokines and increases the production of reactive oxygen species (ROS) in uroepithelial T24 cells. The antioxidant N-acetyl-L-cysteine counteracts these phenomena, a fact which suggests a role for ROS-mediated signaling in CNF1-induced proinflammatory cytokine production.

MeSH terms

  • Bacterial Toxins / toxicity*
  • Cell Line
  • Cytokines / biosynthesis*
  • Cytotoxins / toxicity*
  • Escherichia coli Proteins*
  • Humans
  • Reactive Oxygen Species*
  • Transcription, Genetic / drug effects*
  • Urinary Bladder / drug effects*
  • Urinary Bladder / metabolism

Substances

  • Bacterial Toxins
  • Cytokines
  • Cytotoxins
  • Escherichia coli Proteins
  • Reactive Oxygen Species
  • cytotoxic necrotizing factor type 1