Lymph node blast crisis in chronic myeloid leukemia mimicking T-immunoblastic lymphoma

Haematologica. 1992 Jul-Aug;77(4):311-4.

Abstract

Background: Chronic myeloid leukemia arises from a somatic mutation in a pluripotent stem cell. It generally terminates with a blastic crisis (BC). One third of BC are lymphoid, and most have a pre-B phenotype. Few cases of T-lymphoid BC have been reported. Here we describe a lymph node blast crisis mimicking T-immunoblastic lymphoma.

Methods: Bone marrow and lymph nodes were histologically examined by standard methods and by an immunoperoxidase technique. Cytogenetic studies were also performed on lymph node and blood cells. Analysis of T-cell receptor genes and BCR rearrangements were performed on DNA extracted from both frozen bone marrow and lymph-node cells.

Results: Lymph-node histology showed an infiltration by large lymphoid blasts, consistent with a diagnosis of immunoblastic lymphoma. Blast cells were CD2, CD7, TDT positive, and negative for myeloid and mature lymphoid antigens. The Ph1 chromosome was found in both bone marrow and lymph-node cells. BCR rearrangement was found in the DNA from both bone marrow and lymph-node cells. TCR genes were not rearranged.

Discussion: The present study provides strong evidence that the lymph-node blast crisis of CML can assume the morphological appearance of immunoblastic lymphoma and may retain the immunological phenotype and genetic features of early T cells with BCR rearrangements.

Publication types

  • Case Reports

MeSH terms

  • Antigens, Neoplasm / analysis
  • Biomarkers, Tumor / analysis
  • Blast Crisis / diagnosis*
  • Blast Crisis / pathology
  • Bone Marrow / pathology
  • Fusion Proteins, bcr-abl / genetics
  • Gene Rearrangement, T-Lymphocyte
  • Humans
  • Immunophenotyping
  • Leukemia, Myelogenous, Chronic, BCR-ABL Positive / pathology*
  • Lymph Nodes / pathology*
  • Lymphoma, Large-Cell, Immunoblastic / diagnosis*
  • Lymphoma, T-Cell / diagnosis*
  • Male
  • Middle Aged

Substances

  • Antigens, Neoplasm
  • Biomarkers, Tumor
  • Fusion Proteins, bcr-abl