Myocyte enhancer factor 2 mediates calcium-dependent transcription of the interleukin-2 gene in T lymphocytes: a calcium signaling module that is distinct from but collaborates with the nuclear factor of activated T cells (NFAT)

J Biol Chem. 2004 Apr 9;279(15):14477-80. doi: 10.1074/jbc.C300487200. Epub 2004 Jan 13.

Abstract

The second messenger calcium plays an essential role in the T cell receptor-mediated signal transduction pathways leading to transcription of the interleukin-2 gene. A key mechanism of calcium signaling has been shown to be mediated by calcineurin and NFAT. We report herein that the transcription factor myocyte enhancer factor (MEF)-2 is another calcium signal transducer involved in the regulation of the interleukin (IL)-2 promoter. A MEF2-binding site was identified in proximity to the TATA box of the IL-2 promoter. This site was shown to be bound by MEF2 in both resting and activated T cells. Overexpression of MEF2 enhanced, while overexpression of a dominant negative form of MEF2 or the MEF2-specific transcriptional corepressors Cabin1 and histone deacetylase 4 inhibited, the T cell receptor-dependent activation of an IL-2 reporter gene. Down-regulation of MEF2 by RNA interference in primary human T cells led to the inhibition of endogenous IL-2 transcription. These results suggest that MEF2 is required for the transcriptional activation of IL-2 and likely other cytokine genes in response to calcium signaling and may serve as a novel target for development of immunosuppressants.

Publication types

  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Adaptor Proteins, Signal Transducing
  • Animals
  • Binding Sites
  • Calcineurin / metabolism
  • Calcium / metabolism*
  • Cells, Cultured
  • Chromatin / metabolism
  • DNA-Binding Proteins / physiology*
  • Down-Regulation
  • Genes, Dominant
  • Genes, Reporter
  • Histone Deacetylases / metabolism
  • Humans
  • Immunosuppressive Agents / pharmacology
  • Interleukin-2 / biosynthesis*
  • Interleukin-2 / genetics
  • Interleukin-2 / metabolism
  • Intracellular Signaling Peptides and Proteins
  • Jurkat Cells
  • MEF2 Transcription Factors
  • Mice
  • Myogenic Regulatory Factors
  • Phosphoproteins / metabolism
  • Precipitin Tests
  • Promoter Regions, Genetic
  • Protein Binding
  • RNA Interference
  • RNA, Small Interfering / metabolism
  • Receptors, Antigen, T-Cell / agonists
  • Repressor Proteins / metabolism
  • Signal Transduction
  • T-Lymphocytes / metabolism*
  • Transcription Factors / physiology*
  • Transcription, Genetic*
  • Transcriptional Activation

Substances

  • Adaptor Proteins, Signal Transducing
  • CABIN1 protein, human
  • Cabin1 protein, mouse
  • Chromatin
  • DNA-Binding Proteins
  • Immunosuppressive Agents
  • Interleukin-2
  • Intracellular Signaling Peptides and Proteins
  • MEF2 Transcription Factors
  • Myogenic Regulatory Factors
  • Phosphoproteins
  • RNA, Small Interfering
  • Receptors, Antigen, T-Cell
  • Repressor Proteins
  • Transcription Factors
  • Calcineurin
  • HDAC4 protein, human
  • Histone Deacetylases
  • Calcium