Increased expression of CCL20 in human inflammatory bowel disease

J Clin Immunol. 2004 Jan;24(1):74-85. doi: 10.1023/B:JOCI.0000018066.46279.6b.

Abstract

Inflammatory bowel disease (IBD) constituting Crohn's disease (CD) and ulcerative colitis (UC) is related to a dysregulated T cell response. CCL20 attracts memory T lymphocytes and dendritic cells. We asked whether CCL20 expression is altered in IBD. Colonic biopsies were obtained from 114 subjects with IBD, non-IBD colitis, irritable bowel syndrome, and healthy controls. CCL20 and CCR6 mRNA expression was measured by Taqman-PCR, and protein secretion from colonic explant cultures (CEC) and its regulation by TNF-alpha by ELISA. CCL20, CCR6, and Langerin were identified by immunohistochemistry and immunofluorescence. CCL20 mRNA and protein were severalfold increased in involved CD and UC but not in non-IBD colitis. TNF-alpha increased and anti-TNF-alpha decreased CCL20 release in healthy control CEC but not in involved IBD colonic specimens. CCL20 localized to follicle-associated epithelium, and CCR6 to the adjacent mantle zone of lymphoid follicles. Furthermore, abundant numbers of Langerin(+) immature dendritic cells were identified in the subepithelial space of IBD specimens. CCL20 might regulate the attraction of T lymphocytes and dendritic cells in IBD.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adult
  • Antibodies / immunology
  • Antibodies / metabolism
  • Antigens, CD
  • Antigens, Surface / immunology
  • Antigens, Surface / metabolism
  • Biopsy
  • Chemokine CCL20
  • Chemokines, CC / genetics
  • Chemokines, CC / immunology*
  • Chemokines, CC / metabolism
  • Colitis, Ulcerative / genetics
  • Colitis, Ulcerative / immunology*
  • Colitis, Ulcerative / metabolism
  • Colitis, Ulcerative / pathology
  • Crohn Disease / genetics
  • Crohn Disease / immunology*
  • Crohn Disease / metabolism
  • Crohn Disease / pathology
  • Dendritic Cells / immunology
  • Dendritic Cells / metabolism
  • Epithelium / immunology
  • Epithelium / metabolism
  • Epithelium / pathology
  • Female
  • Humans
  • Lectins, C-Type / immunology
  • Lectins, C-Type / metabolism
  • Macrophage Inflammatory Proteins / genetics
  • Macrophage Inflammatory Proteins / immunology*
  • Macrophage Inflammatory Proteins / metabolism
  • Male
  • Mannose-Binding Lectins / immunology
  • Mannose-Binding Lectins / metabolism
  • Middle Aged
  • RNA, Messenger / metabolism
  • Receptors, CCR6
  • Receptors, Chemokine / immunology
  • Receptors, Chemokine / metabolism
  • Tumor Necrosis Factor-alpha / immunology
  • Tumor Necrosis Factor-alpha / metabolism

Substances

  • Antibodies
  • Antigens, CD
  • Antigens, Surface
  • CCL20 protein, human
  • CCR6 protein, human
  • CD207 protein, human
  • Chemokine CCL20
  • Chemokines, CC
  • Lectins, C-Type
  • Macrophage Inflammatory Proteins
  • Mannose-Binding Lectins
  • RNA, Messenger
  • Receptors, CCR6
  • Receptors, Chemokine
  • Tumor Necrosis Factor-alpha