Cellular immunity in breast cancer patients completing taxane treatment

Clin Cancer Res. 2004 May 15;10(10):3401-9. doi: 10.1158/1078-0432.CCR-1016-03.

Abstract

Purpose: A field study of postchemotherapy immune functioning relative to the use of taxanes is reported. Immune responses in breast cancer patients were analyzed as a function of whether patients received taxane as part of their adjuvant chemotherapy.

Experimental design: Immune levels of 227 stage II/III breast cancer patients were measured immediately after surgery prior to chemotherapy and again 12 months later when all chemotherapies had been completed. T-cell blastogenesis and natural killer (NK) cell lysis levels of patients receiving taxanes (n = 55) were compared with levels of patients not receiving taxanes (n = 172).

Results: Regression analyses were conducted. The administration of taxane as part of combination chemotherapy predicted increased T-cell blastogenesis and NK cell cytotoxicity after the conclusion of all chemotherapies. For the Taxane group, average phytohemagglutinin-induced blastogenesis was 37% higher and NK cell cytotoxicity was 39% higher than the values for the No-Taxane group.

Conclusions: Data from group comparisons with appropriate controls in a sizable clinical sample contravene traditional wisdom that taxanes suppress patients' immune cell functions. Problems in generalizing direct-contact laboratory models to the field of cancer treatment are highlighted.

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, Non-P.H.S.
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Antineoplastic Agents, Phytogenic
  • Breast Neoplasms / drug therapy*
  • Breast Neoplasms / immunology*
  • Chemotherapy, Adjuvant
  • Humans
  • Immune System / drug effects*
  • Immunity, Cellular
  • Killer Cells, Natural / metabolism
  • Leukocytes / metabolism
  • Phytohemagglutinins / metabolism
  • Prognosis
  • Regression Analysis
  • T-Lymphocytes / metabolism
  • Taxoids / metabolism
  • Taxoids / therapeutic use*
  • Time Factors

Substances

  • Antineoplastic Agents, Phytogenic
  • Phytohemagglutinins
  • Taxoids