Structure-related oxidative damage in rat brain after acute and chronic electroshock

Neurochem Res. 2004 Sep;29(9):1749-53. doi: 10.1023/b:nere.0000035811.06277.b3.

Abstract

The role of oxidative stress in electroconvulsive therapy-related effects is not well studied. The purpose of this study was to determine oxidative stress parameters in several brain structures after a single electroconvulsive seizure or multiple electroconvulsive seizures. Rats were given either a single electroconvulsive shock or a series of eight electroconvulsive shocks. Brain regions were isolated, and levels of oxidative stress in the brain tissue (cortex, hippocampus, striatum and cerebellum) were measured. We demonstrated a decrease in lipid peroxidation and protein carbonyls in the hippocampus, cerebellum, and striatum several times after a single electroconvulsive shock or multiple electroconvulsive shocks. In contrast, lipid peroxidation increases both after a single electroconvulsive shock or multiple electroconvulsive shocks in cortex. In conclusion, we demonstrate an increase in oxidative damage in cortex, in contrast to a reduction of oxidative damage in hippocampus, striatum, and cerebellum.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Brain / metabolism
  • Brain / pathology*
  • Cerebellum / pathology
  • Cerebral Cortex / pathology
  • Corpus Striatum / pathology
  • Electroshock / adverse effects*
  • Hippocampus / pathology
  • Male
  • Organ Specificity
  • Oxidative Stress
  • Rats
  • Rats, Wistar