Epigenetic regulation of protein tyrosine phosphatases: potential molecular targets for cancer therapy

Cancer Gene Ther. 2005 Aug;12(8):665-72. doi: 10.1038/sj.cgt.7700828.

Abstract

Promoter methylation-mediated silencing is a hallmark of many established tumor suppressor genes. This review focuses on the methylation and suppression of a receptor-type tyrosine phosphatase gene, PTPRO, in a variety of solid and liquid tumors. In addition, PTPRO exhibits many other characteristics of a bona fide tumor suppressor. Reactivation of genes silenced by methylation using inhibitors of DNA methyltransferases and histone deacetylases, and the potential application of PTPRO as a molecular target for cancer therapy have been discussed.

Publication types

  • Research Support, N.I.H., Extramural
  • Research Support, U.S. Gov't, P.H.S.
  • Review

MeSH terms

  • DNA Methylation
  • Epigenesis, Genetic*
  • Gene Silencing
  • Genes, Tumor Suppressor*
  • Genetic Therapy*
  • Humans
  • Leukemia / enzymology
  • Neoplasms / enzymology
  • Neoplasms / genetics
  • Neoplasms / therapy*
  • Promoter Regions, Genetic / genetics
  • Protein Tyrosine Phosphatases / antagonists & inhibitors*
  • Protein Tyrosine Phosphatases / genetics
  • Protein Tyrosine Phosphatases / physiology*
  • Receptor-Like Protein Tyrosine Phosphatases, Class 2

Substances

  • PTPRU protein, human
  • Protein Tyrosine Phosphatases
  • Receptor-Like Protein Tyrosine Phosphatases, Class 2