Transduction of receptor signals by beta-arrestins

Science. 2005 Apr 22;308(5721):512-7. doi: 10.1126/science.1109237.

Abstract

The transmission of extracellular signals to the interior of the cell is a function of plasma membrane receptors, of which the seven transmembrane receptor family is by far the largest and most versatile. Classically, these receptors stimulate heterotrimeric G proteins, which control rates of generation of diffusible second messengers and entry of ions at the plasma membrane. Recent evidence, however, indicates another previously unappreciated strategy used by the receptors to regulate intracellular signaling pathways. They direct the recruitment, activation, and scaffolding of cytoplasmic signaling complexes via two multifunctional adaptor and transducer molecules, beta-arrestins 1 and 2. This mechanism regulates aspects of cell motility, chemotaxis, apoptosis, and likely other cellular functions through a rapidly expanding list of signaling pathways.

Publication types

  • Research Support, U.S. Gov't, P.H.S.
  • Review

MeSH terms

  • Animals
  • Apoptosis
  • Arrestins / chemistry
  • Arrestins / genetics
  • Arrestins / metabolism*
  • Cell Movement
  • Chemotaxis
  • Endocytosis
  • Heterotrimeric GTP-Binding Proteins / metabolism
  • Humans
  • Mitogen-Activated Protein Kinases / metabolism
  • Models, Biological
  • Models, Molecular
  • Protein Conformation
  • Protein-Tyrosine Kinases / metabolism
  • Receptors, G-Protein-Coupled / metabolism*
  • Second Messenger Systems / physiology
  • Signal Transduction*
  • beta-Arrestins

Substances

  • Arrestins
  • Receptors, G-Protein-Coupled
  • beta-Arrestins
  • seven-transmembrane G-protein-coupled receptor
  • Protein-Tyrosine Kinases
  • Mitogen-Activated Protein Kinases
  • Heterotrimeric GTP-Binding Proteins