Oxadiazole derivatives as a novel class of antimitotic agents: Synthesis, inhibition of tubulin polymerization, and activity in tumor cell lines

Bioorg Med Chem Lett. 2006 Mar 1;16(5):1191-6. doi: 10.1016/j.bmcl.2005.11.094. Epub 2006 Jan 11.

Abstract

Oxadiazole derivatives were synthesized and evaluated for their ability to inhibit tubulin polymerization and to cause mitotic arrest in tumor cells. The most potent compounds inhibited tubulin polymerization at concentrations below 1 microM. Lead analogs caused mitotic arrest of A431 human epidermoid cells and cells derived from multi-drug resistant tumors (10, EC(50)=7.8 nM). Competition for the colchicine binding site and pharmacokinetic properties of selected potent compounds were also investigated and are reported herein, along with structure-activity relationships for this novel series of antimitotic agents.

MeSH terms

  • Animals
  • Antimitotic Agents / chemical synthesis*
  • Antimitotic Agents / chemistry
  • Antimitotic Agents / classification
  • Antimitotic Agents / pharmacology*
  • Biopolymers / chemistry
  • Biopolymers / metabolism
  • Cell Line, Tumor
  • Humans
  • Inhibitory Concentration 50
  • Mice
  • Molecular Structure
  • Oxadiazoles / chemical synthesis
  • Oxadiazoles / chemistry*
  • Oxadiazoles / classification
  • Oxadiazoles / pharmacology*
  • Protein Conformation / drug effects
  • Structure-Activity Relationship
  • Tubulin / chemistry*
  • Tubulin / metabolism*

Substances

  • Antimitotic Agents
  • Biopolymers
  • Oxadiazoles
  • Tubulin