Over-expression of c-Myb increases the frequency of hemogenic precursors in the endothelial cell population

Genes Cells. 2006 Aug;11(8):859-70. doi: 10.1111/j.1365-2443.2006.00985.x.

Abstract

Definitive hematopoiesis has been proposed to arise from hemogenic endothelial cells during mouse embryogenesis. The c-myb proto-oncogene is essential for the development of definitive hematopoiesis and was reported to be activated in hemogenic endothelial cells. To investigate whether c-Myb is involved in regulating the development of hemogenic endothelial cells, we conditionally induced c-myb over-expression during the in vitro differentiation of embryonic stem cells. VE-cadherin+ CD45- cells inducibly expressing c-Myb showed an increase in multilineage colony formation as well as an augmented capacity of the colony forming cells to self-renew in vitro under the condition that only the endogenous c-myb gene was expressed during differentiation of hematopoietic cells. Over-expression of c-Myb in the endothelial population led to activation of genes associated with definitive hematopoiesis such as Runx1, Hoxb4, Mll and Etv6. Our data provide evidence that c-Myb is able to exert an effect in endothelial cells which fosters the establishment of their hemogenic potential.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • B-Lymphocytes / metabolism
  • Cell Death
  • Cell Differentiation
  • Cell Proliferation
  • Cells, Cultured
  • Embryonic Induction / genetics
  • Endothelial Cells / physiology*
  • Gene Expression Regulation
  • Genes, myb / physiology*
  • Hematopoiesis / genetics
  • Hematopoietic Stem Cells / metabolism
  • Hematopoietic Stem Cells / physiology*
  • Mice
  • Transfection