Accumulation of class switched IgD-IgM- memory B cells in the cerebrospinal fluid during neuroinflammation

J Neuroimmunol. 2006 Nov;180(1-2):33-9. doi: 10.1016/j.jneuroim.2006.06.031. Epub 2006 Sep 6.

Abstract

Inflammatory diseases of the central nervous system (CNS) are characterized by cerebrospinal fluid (CSF) pleocytosis often involving the recruitment of B cells. Little is still known about B cells that are found in the CSF during neuroinflammation. To address the phenotype of these B cells, we studied the distribution of the major B cell subsets in peripheral blood (PB) and CSF of 25 patients with inflammatory diseases of the nervous system by flow cytometry. Six different B cell subsets were identified in PB and CSF according to the surface expression of IgM, IgD, CD27 and CD19. In all patients analysed, memory B cells outnumbered naïve B cells in the CSF, whereas naïve B cells were more prevalent in PB. The accumulation of memory B cells in the CSF was largely due to the recruitment of IgM-IgD- class switched memory B cells. The distribution of IgM+IgD+, IgM-IgD+, IgM+IgD- memory cells and immature cells did not differ significantly between CSF and PB. These findings demonstrate a selective recruitment of IgM-IgD- memory B cells to the CSF suggesting a specific role of these cells during neuroinflammation.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adult
  • Aged
  • Antibody Formation / immunology
  • Antigens, CD19 / immunology
  • Autoantibodies / immunology
  • B-Lymphocytes / immunology*
  • Cell Differentiation / immunology
  • Cerebrospinal Fluid / cytology
  • Cerebrospinal Fluid / immunology*
  • Encephalitis / cerebrospinal fluid
  • Encephalitis / immunology*
  • Encephalitis / physiopathology
  • Female
  • Humans
  • Immunoglobulin Class Switching / immunology
  • Immunoglobulin D / immunology*
  • Immunoglobulin M / immunology*
  • Immunologic Memory / immunology*
  • Male
  • Middle Aged
  • Tumor Necrosis Factor Receptor Superfamily, Member 7 / immunology

Substances

  • Antigens, CD19
  • Autoantibodies
  • Immunoglobulin D
  • Immunoglobulin M
  • Tumor Necrosis Factor Receptor Superfamily, Member 7