Flow cytometric analysis of phosphorylated histone H2AX following exposure to ionizing radiation in human microvascular endothelial cells

J Radiat Res. 2006 Nov;47(3-4):245-57. doi: 10.1269/jrr.0628. Epub 2006 Sep 6.

Abstract

We applied a flow cytometric method to quantify IR-induced histone H2AX phosphorylation at serine 139 (gammaH2AX) and compared those values to those obtained using a standard microscopy based foci counting method. After PFA fixation, methanol permeabilization was suitable for both FITC- or Alexa647-gammaH2AX. In contrast, Alexa647-gammaH2AX was not suitable for ethanol permeabilization. Antibody concentrations at 1-2 microg/ml yielded the highest gammaH2AX positive percentage for both antibodies. Without DAPI staining, gammaH2AX formation can be measured as a relative fold increase. Values determined by bivariant flow cytometric analysis and those obtained using microscopic foci formation exhibited a good quantitative correlation. Values obtained by both methods could vary according to the gating or threshold setting used. gammaH2AX positive cells increased as a function of radiation dose (2-16 Gy) followed by a dose-dependent decay. The free radical scavenger N-acetyl-L-cysteine (NAC), if administered at a concentration of 4 mM 30 min before IR, was effective in reducing IR-induced gammaH2AX formation in all phases of the cell cycle. We have developed a simplified and quantitative flow cytometry based method to measure IR-induced gammaH2AX in cells and demonstrated strong correlation to values obtained by a standard automated digital microscopic foci analysis along with NIH ImageJ custom macro software.

Publication types

  • Research Support, N.I.H., Extramural
  • Research Support, U.S. Gov't, Non-P.H.S.

MeSH terms

  • Cells, Cultured
  • DNA / radiation effects*
  • DNA Breaks*
  • Dose-Response Relationship, Radiation
  • Endothelial Cells / physiology*
  • Endothelial Cells / radiation effects*
  • Flow Cytometry / methods*
  • Histones / genetics*
  • Histones / radiation effects*
  • Histones / ultrastructure
  • Humans
  • Microcirculation / cytology
  • Microcirculation / physiology
  • Microcirculation / radiation effects
  • Radiation Dosage
  • Radiation, Ionizing

Substances

  • H2AX protein, human
  • Histones
  • DNA