Down-regulation of Sox10 with specific small interfering RNA promotes transdifferentiation of Schwannoma cells into myofibroblasts

Differentiation. 2006 Dec;74(9-10):542-51. doi: 10.1111/j.1432-0436.2006.00084.x.

Abstract

Neural crest-derived cells are noted for the long lasting plasticity during lineage commitment process and the potential for transdifferentiation into other neural crest derivatives. Schwann cells in particular have been reported to transdifferentiate into melanocytes and myofibroblasts. Detailed studies of transdifferentiation at the molecular level have been hampered by difficulty in isolating sufficient quantity of primary cells or cellular materials. Here, we describe a robust in vitro system in which Schwannoma cells undergo an apparent transdifferentiation into myofibroblasts. Importantly, we induce the transdifferentiation by down-regulating a single transcription factor, Sox10, thereby identifying a key molecular event in this process. Myofibroblasts thus generated showed carbachol-stimulated contraction and calcium transients and express several established myofibroblast-specific genes. These results suggest that generating desired cell types based on "knock-down" of critical genes may be a viable strategy.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Actins / analysis
  • Actins / metabolism
  • Animals
  • Carbachol / pharmacology
  • Cell Differentiation
  • Cells, Cultured
  • DNA-Binding Proteins / antagonists & inhibitors*
  • DNA-Binding Proteins / genetics
  • DNA-Binding Proteins / metabolism
  • Down-Regulation*
  • Fibroblasts / cytology*
  • Fibroblasts / metabolism
  • High Mobility Group Proteins / antagonists & inhibitors*
  • High Mobility Group Proteins / genetics
  • High Mobility Group Proteins / metabolism
  • Immunohistochemistry
  • Neuroglia / cytology
  • Phenotype
  • RNA Interference*
  • RNA, Small Interfering / metabolism
  • Rats
  • SOXE Transcription Factors
  • Schwann Cells / cytology*
  • Schwann Cells / metabolism
  • Transcription Factors / antagonists & inhibitors*
  • Transcription Factors / genetics
  • Transcription Factors / metabolism
  • Transfection

Substances

  • Actins
  • DNA-Binding Proteins
  • High Mobility Group Proteins
  • RNA, Small Interfering
  • SOXE Transcription Factors
  • Sox10 protein, rat
  • Transcription Factors
  • smooth muscle actin, rat
  • Carbachol