Monocytes may amplify their recruitment into inflammatory lesions by inducing monocyte chemotactic protein

Arterioscler Thromb. 1992 Jan;12(1):78-82. doi: 10.1161/01.atv.12.1.78.

Abstract

By Northern analysis, freshly isolated monocytes contained no detectable mRNA for monocyte chemotactic protein-1 (MCP-1). However, after 4 hours of incubation at 37 degrees C, MCP-1 mRNA was clearly induced in the monocytes and was found to be highly dependent and directly proportional to the monocyte density. The level of MCP-1 mRNA continued to increase, reaching a peak after 22 hours of incubation. After 3 days in culture, MCP-1 mRNA levels had declined substantially and after 8 days were undetectable in the monocytes/macrophages. The amount of MCP-1 protein secreted correlated with the density-dependent increase in MCP-1 message. We hypothesize that the migration of monocytes into inflammatory lesions may be amplified by the density and time-dependent induction of MCP-1.

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Base Sequence
  • Cell Adhesion
  • Cell Count
  • Cell Differentiation
  • Cells, Cultured
  • Chemokine CCL2
  • Chemotactic Factors / biosynthesis*
  • Chemotactic Factors / genetics
  • DNA Probes / chemistry
  • Humans
  • Inflammation / pathology*
  • Molecular Sequence Data
  • Monocytes / pathology
  • Monocytes / physiology*
  • RNA, Messenger / biosynthesis

Substances

  • Chemokine CCL2
  • Chemotactic Factors
  • DNA Probes
  • RNA, Messenger