Estrogen receptor-alpha variants increase risk of Alzheimer's disease in women with Down syndrome

Dement Geriatr Cogn Disord. 2008;25(5):476-82. doi: 10.1159/000126495. Epub 2008 Apr 14.

Abstract

Background: Genetic variants that affect estrogen activity may influence the risk of Alzheimer's disease (AD). Two tightly linked polymorphisms (PvuII and XbaI) in the first intron of estrogen receptor 1 (ESR1), the gene for ER-alpha, have been reported to influence estrogen receptor expression and may influence the risk of AD.

Methods: We examined the relation of polymorphisms in ESR1 to the risk of AD in women with Down syndrome. The subjects (181 women with DS, 41-78 years of age) were followed at 14- to 18-month intervals. Information from cognitive assessments, caregiver interviews, medical record reviews and neurological examinations was used to classify dementia. Genomic DNA was genotyped for 5 single-nucleotide polymorphisms in the upstream region and the first exon/intron of the ESR1 gene. Their association with dementia risk was evaluated, adjusting for covariates.

Results: Women with at least 1 copy of the C allele at rs2234693 (PvuII) and those homozygous for the C allele at rs2077647 had an almost 3-fold increase in the risk of AD, compared with women without the C allele. The increased risks were independent of the apolipoprotein E genotype.

Conclusion: These findings support a role for estrogen receptor activity in the development of AD in women with Down syndrome.

MeSH terms

  • Adult
  • Aged
  • Alzheimer Disease / epidemiology*
  • Alzheimer Disease / genetics*
  • Apolipoprotein E4 / genetics
  • Down Syndrome / epidemiology*
  • Down Syndrome / genetics*
  • Estrogen Receptor alpha / genetics*
  • Female
  • Gene Frequency
  • Genetic Predisposition to Disease / epidemiology
  • Genotype
  • Humans
  • Middle Aged
  • Polymorphism, Single Nucleotide
  • Risk Factors

Substances

  • Apolipoprotein E4
  • Estrogen Receptor alpha