Preventive and curative effects of cyclophosphamide in an animal model of Guillain Barrè syndrome

J Neuroimmunol. 2008 May 30;196(1-2):107-15. doi: 10.1016/j.jneuroim.2008.03.008. Epub 2008 May 2.

Abstract

The immunosuppressive agent cyclophosphamide (CY) was tested in rat experimental allergic neuritis (EAN), a preclinical model of Guillain Barrè syndrome (GBS). CY prophylaxis (day 0 and 14 post-immunization [p.i.]) effectively prevents clinical and histological signs of EAN and also reduces the cytokine and the NF-kappaB p65 expression in the nervous tissue. When administered therapeutically (day 14th p.i.) to rats with established disease CY only affects the clinical symptoms. Both the prophylactic and therapeutic treatment with CY reduced ex vivo antigen-specific T cell proliferative responses. These results warrant studies with CY in those cases of GBS resistant to conventional therapies.

MeSH terms

  • Animals
  • Cell Proliferation / drug effects
  • Cyclophosphamide / therapeutic use*
  • Cytokines / metabolism
  • Disease Models, Animal
  • Drug Administration Schedule
  • Guillain-Barre Syndrome / chemically induced
  • Guillain-Barre Syndrome / drug therapy*
  • Guillain-Barre Syndrome / metabolism
  • Guillain-Barre Syndrome / pathology
  • Guillain-Barre Syndrome / prevention & control*
  • Immunosuppressive Agents / therapeutic use*
  • Male
  • Myelin P0 Protein
  • Rats
  • Sciatic Nerve / drug effects
  • Sciatic Nerve / metabolism
  • Statistics, Nonparametric
  • Time Factors
  • eIF-2 Kinase / metabolism

Substances

  • Cytokines
  • Immunosuppressive Agents
  • Myelin P0 Protein
  • Cyclophosphamide
  • eIF-2 Kinase