Angiotensin-converting enzyme insertion/deletion polymorphism in patients with acute and chronic pancreatitis

Eur J Gastroenterol Hepatol. 2009 Sep;21(9):1032-5. doi: 10.1097/MEG.0b013e328326f586.

Abstract

Background: Reduction in angiotensin-converting enzyme (ACE) activity has been shown to attenuate pancreatic stellate cell activation and pancreatic fibrosis and suggested as a potential treatment for chronic pancreatitis. The ACE gene insertion/deletion (I/D) polymorphism in intron 16 accounts for nearly half the variation in serum ACE levels. This study determined the frequency of the I/D polymorphism in patients with acute and chronic pancreatitis.

Methods: In total, 887 patients (346 with alcoholic, 443 with nonalcoholic, and 98 with acute pancreatitis) were enrolled, and were compared with 1294 healthy controls. Genotyping of the I/D polymorphism was performed by PCR or melting curve analyses.

Results: No significant differences were found in the prevalence of the ACE-deletion genotype frequencies when patients with alcoholic (27.5%), nonalcoholic (26.4%), and acute pancreatitis (32.7%) were compared with controls (26.9%). Likewise, allele frequencies of the ACE deletion polymorphism were not significantly different in patients with alcoholic (53.8%), nonalcoholic (50.6%), and acute pancreatitis (54.1%) and controls (52.7%).

Conclusion: The I/D polymorphism of the ACE gene was not found to be associated with acute and chronic pancreatitis.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Acute Disease
  • Adolescent
  • Adult
  • Aged
  • Aged, 80 and over
  • Child
  • Child, Preschool
  • Disease Susceptibility
  • Female
  • Finnland
  • Gene Deletion*
  • Genotype
  • Deutschland
  • Humans
  • Male
  • Middle Aged
  • Mutagenesis, Insertional / genetics*
  • Pancreatitis / genetics*
  • Pancreatitis / metabolism
  • Pancreatitis, Chronic / genetics*
  • Pancreatitis, Chronic / metabolism
  • Peptidyl-Dipeptidase A / genetics*
  • Peptidyl-Dipeptidase A / metabolism
  • Polymorphism, Genetic*
  • Young Adult

Substances

  • Peptidyl-Dipeptidase A