Marked downregulation of the granulopoiesis regulator LEF1 is associated with disease progression in the myelodysplastic syndromes

Br J Haematol. 2009 Jun;146(1):86-90. doi: 10.1111/j.1365-2141.2009.07720.x. Epub 2009 May 5.

Abstract

Lymphoid enhancer-binding factor 1 (LEF1) is a neutrophilic granulopoiesis regulator whose absence is critical in congenital neutropenia. We have shown LEF1 downregulation in the CD34(+) cells of the majority of myelodysplastic syndromes (MDS) patients. LEF1 was the most significant differentially expressed gene between early and advanced MDS. Marked LEF1 downregulation was found in 27/32 patients with advanced MDS and in 6/35 patients with early MDS, and was associated with neutropenia. Downregulation of LEF1 mRNA was reflected at the protein level. Immunostaining for CD34/LEF1 may represent a marker of advanced MDS. LEF1 may play a role in the defective maturation of myeloid progenitors in MDS.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Antigens, CD34 / analysis
  • Case-Control Studies
  • Disease Progression
  • Down-Regulation
  • Gene Expression Profiling / methods
  • Humans
  • Immunohistochemistry
  • Lymphoid Enhancer-Binding Factor 1 / analysis
  • Lymphoid Enhancer-Binding Factor 1 / genetics*
  • Myelodysplastic Syndromes / genetics*
  • Myelodysplastic Syndromes / immunology
  • Myelodysplastic Syndromes / metabolism
  • Neutropenia
  • Oligonucleotide Array Sequence Analysis
  • RNA, Messenger / analysis*
  • Reverse Transcriptase Polymerase Chain Reaction
  • Sequence Analysis, DNA

Substances

  • Antigens, CD34
  • Lymphoid Enhancer-Binding Factor 1
  • RNA, Messenger