Lack of association with rheumatoid arthritis of selected polymorphisms in 4 candidate genes: CFH, CD209, eotaxin-3, and MHC2TA

J Rheumatol. 2009 Aug;36(8):1590-5. doi: 10.3899/jrheum.090022. Epub 2009 Jun 30.

Abstract

Objective: To investigate associations with rheumatoid arthritis (RA) of single-nucleotide polymorphisms (SNP) in 4 candidate genes, complement factor H (CFH), CD209 or DC-SIGN, eotaxin-3, and the MHC class II Transactivator (MHC2TA) genes. These SNP have been reported as important for RA (eotaxin-3 and MHC2TA) or for other immune-mediated diseases (CFH and CD209).

Methods: Genotypes for the 7 selected SNP were obtained from 1587 patients with RA and 1570 controls of Spanish ancestry. Analyses were carried out after stratification for sex, erosions, rheumatoid factor, shared epitope, anti-cyclic citrullinated peptide antibodies, and the R620W PTPN22 SNP.

Results: None of the comparisons between patients with RA and controls or between the different strata of patients according to disease features was significant.

Conclusion: None of the SNP in CFH and CD209 showed evidence of association with RA. We did not replicate the association of eotaxin-3 with RA described in Koreans, or that of the MHC2T SNP.

MeSH terms

  • Adult
  • Arthritis, Rheumatoid / genetics*
  • Case-Control Studies
  • Cell Adhesion Molecules / genetics*
  • Chemokine CCL26
  • Chemokines, CC / genetics*
  • Complement Factor H / genetics
  • Female
  • Gene Frequency
  • Genetic Predisposition to Disease
  • Genotype
  • Humans
  • Lectins, C-Type / genetics*
  • Male
  • Middle Aged
  • Nuclear Proteins / genetics*
  • Polymorphism, Single Nucleotide*
  • Receptors, Cell Surface / genetics*
  • Spanien
  • Trans-Activators / genetics*

Substances

  • CCL26 protein, human
  • CFH protein, human
  • Cell Adhesion Molecules
  • Chemokine CCL26
  • Chemokines, CC
  • DC-specific ICAM-3 grabbing nonintegrin
  • Lectins, C-Type
  • MHC class II transactivator protein
  • Nuclear Proteins
  • Receptors, Cell Surface
  • Trans-Activators
  • Complement Factor H