iPS programmed without c-MYC yield proficient cardiogenesis for functional heart chimerism

Circ Res. 2009 Sep 25;105(7):648-56. doi: 10.1161/CIRCRESAHA.109.203109. Epub 2009 Aug 20.

Abstract

Rationale: Induced pluripotent stem cells (iPS) allow derivation of pluripotent progenitors from somatic sources. Originally, iPS were induced by a stemness-related gene set that included the c-MYC oncogene.

Objective: Here, we determined from embryo to adult the cardiogenic proficiency of iPS programmed without c-MYC, a cardiogenicity-associated transcription factor.

Methods and results: Transgenic expression of 3 human stemness factors SOX2, OCT4, and KLF4 here reset murine fibroblasts to the pluripotent ground state. Transduction without c-MYC reversed cellular ultrastructure into a primitive archetype and induced stem cell markers generating 3-germ layers, all qualifiers of acquired pluripotency. Three-factor induced iPS (3F-iPS) clones reproducibly demonstrated cardiac differentiation properties characterized by vigorous beating activity of embryoid bodies and robust expression of cardiac Mef2c, alpha-actinin, connexin43, MLC2a, and troponin I. In vitro isolated iPS-derived cardiomyocytes demonstrated functional excitation-contraction coupling. Chimerism with 3F-iPS derived by morula-stage diploid aggregation was sustained during prenatal heart organogenesis and contributed in vivo to normal cardiac structure and overall performance in adult tumor-free offspring.

Conclusions: Thus, 3F-iPS bioengineered without c-MYC achieve highest stringency criteria for bona fide cardiogenesis enabling reprogrammed fibroblasts to yield de novo heart tissue compatible with native counterpart throughout embryological development and into adulthood.

Publication types

  • Research Support, N.I.H., Extramural
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Actinin / metabolism
  • Action Potentials
  • Animals
  • Calcium Signaling
  • Cell Lineage
  • Cell Transdifferentiation* / genetics
  • Cells, Cultured
  • Chimerism
  • Connexin 43 / metabolism
  • Embryo Culture Techniques
  • Female
  • Fibroblasts / metabolism*
  • Fibroblasts / ultrastructure
  • Gene Expression Regulation, Developmental
  • Humans
  • Kruppel-Like Factor 4
  • Kruppel-Like Transcription Factors / genetics
  • Kruppel-Like Transcription Factors / metabolism*
  • MEF2 Transcription Factors
  • Mice
  • Mice, Inbred C57BL
  • Mice, Nude
  • Myocardial Contraction* / genetics
  • Myocytes, Cardiac / metabolism*
  • Myocytes, Cardiac / ultrastructure
  • Myogenic Regulatory Factors / metabolism
  • Myosin Light Chains / metabolism
  • Octamer Transcription Factor-3 / genetics
  • Octamer Transcription Factor-3 / metabolism*
  • Organogenesis
  • Pluripotent Stem Cells / metabolism*
  • Pluripotent Stem Cells / ultrastructure
  • Pregnancy
  • Proto-Oncogene Proteins c-myc / metabolism
  • SOXB1 Transcription Factors / genetics
  • SOXB1 Transcription Factors / metabolism*
  • Tissue Engineering / methods
  • Transduction, Genetic
  • Troponin I / metabolism

Substances

  • Connexin 43
  • GJA1 protein, mouse
  • KLF4 protein, human
  • Klf4 protein, mouse
  • Kruppel-Like Factor 4
  • Kruppel-Like Transcription Factors
  • MEF2 Transcription Factors
  • Mef2c protein, mouse
  • Mlc2a protein, mouse
  • Myogenic Regulatory Factors
  • Myosin Light Chains
  • Octamer Transcription Factor-3
  • POU5F1 protein, human
  • Proto-Oncogene Proteins c-myc
  • SOX2 protein, human
  • SOXB1 Transcription Factors
  • Troponin I
  • Actinin