Effect of dietary conjugated linoleic acid supplementation on early inflammatory responses during cutaneous wound healing

Mediators Inflamm. 2010:2010:342328. doi: 10.1155/2010/342328. Epub 2010 Aug 17.

Abstract

Inflammatory response is considered the most important period that regulates the entire healing process. Conjugated linoleic acid (CLA), a class of linoleic acid positional and geometric isomers, is well known for its antioxidant and anti-inflammatory properties. We hypothesized that dietary CLA supplementation accelerates cutaneous wound healing by regulating antioxidant and anti-inflammatory functions. To investigate wound closure rates and inflammatory responses, we used a full-thickness excisional wound model after 2-week treatments with control, 0.5%, or 1% CLA-supplemented diet. Mice fed dietary CLA supplementation had reduced levels of oxidative stress and inflammatory markers. Moreover, the wound closure rate was improved significantly in mice fed a 1% CLA-supplemented diet during early stage of wound healing (inflammatory stage). We conclude that dietary CLA supplementation enhances the early stage of cutaneous wound healing as a result of modulating oxidative stress and inflammatory responses.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Body Weight / drug effects
  • Cyclooxygenase 2 / metabolism
  • Dietary Supplements*
  • Eating / drug effects
  • Female
  • Heme Oxygenase-1 / metabolism
  • Inflammation / metabolism
  • Inflammation / pathology*
  • Linoleic Acids, Conjugated* / administration & dosage
  • Linoleic Acids, Conjugated* / pharmacology
  • Liver / drug effects
  • Liver / metabolism
  • Malondialdehyde / metabolism
  • Mice
  • Oxidative Stress
  • Reactive Oxygen Species / metabolism
  • Skin* / drug effects
  • Skin* / pathology
  • Superoxide Dismutase / metabolism
  • Wound Healing / drug effects*

Substances

  • Linoleic Acids, Conjugated
  • Reactive Oxygen Species
  • Malondialdehyde
  • Heme Oxygenase-1
  • Ptgs2 protein, mouse
  • Cyclooxygenase 2
  • Superoxide Dismutase