E-cadherin and alpha-catenin expression in normal, hyperplastic and neoplastic endometrium

Anticancer Res. 2010 Dec;30(12):4993-7.

Abstract

The aim of this study was to determine whether modulation of expression of cell adhesion molecules may occur in neoplastic transformation of endometrial epithelium.

Materials and methods: E-Cadherin and α-catenin protein expression were evaluated by immunohistochemistry in 124 biopsies representative of normal, hyperplastic and neoplastic endometrium.

Results: In normal endometrium (proliferative, secretive and atrophic endometrium) strong homogeneous, E-cadherin and α-catenin reactivity was found; 58.3% and 66.6% of biopsies representative of simple hyperplastic endometrium were homogeneously positive for E-cadherin and α-catenin, respectively, whereas no samples representative of atypical hyperplasia showed evidence of homogeneous E-cadherin or α-catenin expression. No expression of homogeneous E-cadherin was seen in endometrial adenocarcinomas; α-catenin homogeneous immunostaining was observed in 2 G1 and 2 G2 out of 22 adenocarcinoma samples (18.2%). A homogeneous co-expression of both molecules was seen only in normal (70%) and simple hyperplastic (46%) endometrium.

Conclusion: These results suggest that E-cadherin and α-catenin down-regulation might be associated with neoplastic transformation of endometrial tissues.

MeSH terms

  • Adenocarcinoma / metabolism
  • Cadherins / biosynthesis*
  • Cadherins / metabolism
  • Endometrial Hyperplasia / metabolism*
  • Endometrial Neoplasms / metabolism*
  • Endometrium / metabolism
  • Female
  • Humans
  • Immunohistochemistry
  • Prognosis
  • Tissue Distribution
  • alpha Catenin / biosynthesis*
  • alpha Catenin / metabolism

Substances

  • Cadherins
  • alpha Catenin