miR-126 inhibits proliferation of small cell lung cancer cells by targeting SLC7A5

FEBS Lett. 2011 Apr 20;585(8):1191-6. doi: 10.1016/j.febslet.2011.03.039. Epub 2011 Mar 23.

Abstract

Despite intensive efforts to improve therapies, small cell lung cancer (SCLC) still has a dismal median survival of 18 months. Since miR-126 is under-expressed in the majority of SCLC tumors, we investigated the effect of miR-126 overexpression on the proliferation and cell cycle distribution of H69 cells. Our results demonstrate that miR-126 inhibits proliferation of H69 cells, by delaying the cells in the G1 phase. Short interfering RNA (siRNA) mediated suppression of SLC7A5, a predicted target of mir-126, has the same effect on H69 cells. We also show for the first time that SLC7A5 is a direct target of miR-126.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Blotting, Western
  • Cell Line, Tumor
  • Cell Proliferation*
  • G1 Phase / genetics
  • Gene Expression Regulation, Neoplastic
  • Humans
  • Immunohistochemistry
  • Large Neutral Amino Acid-Transporter 1 / genetics*
  • Large Neutral Amino Acid-Transporter 1 / metabolism
  • Lung Neoplasms / genetics*
  • Lung Neoplasms / metabolism
  • Lung Neoplasms / pathology
  • MicroRNAs / genetics*
  • RNA Interference
  • Reverse Transcriptase Polymerase Chain Reaction
  • Small Cell Lung Carcinoma / genetics*
  • Small Cell Lung Carcinoma / metabolism
  • Small Cell Lung Carcinoma / pathology
  • Time Factors
  • Tissue Array Analysis

Substances

  • Large Neutral Amino Acid-Transporter 1
  • MIRN126 microRNA, human
  • MicroRNAs