Mitogenic activity of endothelin-1 and -3 on vascular smooth muscle cells is inhibited by atrial natriuretic peptides

Artery. 1990;17(6):311-24.

Abstract

The effect of endothelin-1 (ET-1) and endothelin-3 (ET-3) on the proliferation of aortic vascular smooth muscle cells (VSMC) from spontaneously hypertensive rats (SHR) was assessed by the measurement of (3H)-thymidine incorporation into DNA. ET-1 and ET-3 stimulated the DNA synthesis in a dose-dependent manner. ET-3 was less potent than ET-1 in its stimulatory activity. The increase in (3H)-thymidine incorporation is clearly attenuated by Atriopeptin III (ANP 5-28). Atriopeptin I (ANP 5-25), a ANP fragment with the reduced biological activity compared to ANP 5-28, has no effect on the endothelin mediated stimulation of DNA-synthesis.

MeSH terms

  • Animals
  • Atrial Natriuretic Factor / pharmacology*
  • Dose-Response Relationship, Drug
  • Endothelins / antagonists & inhibitors
  • Endothelins / pharmacology*
  • Mitogens / antagonists & inhibitors
  • Mitogens / pharmacology*
  • Muscle, Smooth, Vascular / cytology
  • Muscle, Smooth, Vascular / drug effects*
  • Muscle, Smooth, Vascular / metabolism
  • Peptide Fragments
  • Rats
  • Rats, Inbred SHR
  • Thymidine / metabolism

Substances

  • Endothelins
  • Mitogens
  • Peptide Fragments
  • atrial natriuretic factor prohormone (103-126)
  • Atrial Natriuretic Factor
  • atrial natriuretic factor prohormone (103-123)
  • Thymidine