Hormonal regulation of complement biosynthesis in human cell lines--II. Upregulation of the biosynthesis of complement components C3, factor B and C1 inhibitor by interleukin-6 and interleukin-1 in human hepatoma cell line

Mol Immunol. 1990 Feb;27(2):197-201. doi: 10.1016/0161-5890(90)90115-g.

Abstract

The effect of interleukin (IL)-6 and IL-1 on the biosynthesis of complement components C3, factor B, C2, C4 and C1 inhibitor (C1 inh), as well as that of albumin, was studied in vitro in human hepatoma-derived cell line, HepG2. Measuring the amounts of secreted complement proteins we detected a significant upregulation of C3 by both hormones. The enhancement of the factor B and especially that of C1 inh production was predominant by IL-6. In our experimental system neither IL-1 nor IL-6 affected the biosynthesis of C2 and C4. Albumin secretion was significantly decreased only in the simultaneous presence of IL-1 and IL-6. Detection of the changes in the amounts of C3- and factor B-specific mRNA of HepG2 cells suggests a pretranslational regulation by these cytokines. The secretion of C3 and factor B was markedly potentiated when IL-1 and IL-6 were added together. However only the gene expression of factor B, but not of C3, was found to reveal synergism. IL-6 enhanced the in vitro production of C3 in mouse hepatocytes as well. This effect was greatly potentiated in the presence of histamine.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Carcinoma, Hepatocellular
  • Cell Line
  • Complement C1 Inactivator Proteins / biosynthesis*
  • Complement C1 Inactivator Proteins / genetics
  • Complement C3 / biosynthesis*
  • Complement C3 / genetics
  • Complement Factor B / biosynthesis*
  • Complement Factor B / genetics
  • Enzyme Precursors / biosynthesis*
  • Gene Expression Regulation
  • Humans
  • Interleukin-1 / pharmacology*
  • Interleukin-6 / pharmacology*
  • Liver Neoplasms
  • Recombinant Proteins / pharmacology
  • Tumor Cells, Cultured
  • Up-Regulation*

Substances

  • Complement C1 Inactivator Proteins
  • Complement C3
  • Enzyme Precursors
  • Interleukin-1
  • Interleukin-6
  • Recombinant Proteins
  • Complement Factor B