Abstract
Endocrine therapies for breast cancer that target the estrogen receptor (ER) are ineffective in the 25%-30% of cases that are ER negative (ER-). Androgen receptor (AR) is expressed in 60%-70% of breast tumors, independent of ER status. How androgens and AR regulate breast cancer growth remains largely unknown. We find that AR is enriched in ER- breast tumors that overexpress HER2. Through analysis of the AR cistrome and androgen-regulated gene expression in ER-/HER2+ breast cancers we find that AR mediates ligand-dependent activation of Wnt and HER2 signaling pathways through direct transcriptional induction of WNT7B and HER3. Specific targeting of AR, Wnt or HER2 signaling impairs androgen-stimulated tumor cell growth suggesting potential therapeutic approaches for ER-/HER2+ breast cancers.
Copyright © 2011 Elsevier Inc. All rights reserved.
Publication types
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Research Support, N.I.H., Extramural
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Research Support, U.S. Gov't, Non-P.H.S.
MeSH terms
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Androgens / pharmacology
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Anilides / pharmacology
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Animals
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Breast Neoplasms / genetics
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Breast Neoplasms / metabolism*
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Breast Neoplasms / pathology
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Cell Line, Tumor
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Cell Proliferation / drug effects
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Dihydrotestosterone / pharmacology
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Female
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Gene Expression Profiling
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Gene Expression Regulation, Neoplastic / drug effects
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Hepatocyte Nuclear Factor 3-alpha / metabolism
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Humans
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Mice
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Nitriles / pharmacology
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Receptor, ErbB-2 / genetics
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Receptor, ErbB-2 / metabolism
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Receptors, Androgen / metabolism*
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Receptors, Estrogen / metabolism*
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Signal Transduction / drug effects
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Signal Transduction / genetics
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Tosyl Compounds / pharmacology
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Transcriptional Activation / drug effects
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Up-Regulation / drug effects
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Wnt Proteins / genetics
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Wnt Proteins / metabolism
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Xenograft Model Antitumor Assays
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beta Catenin / genetics
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beta Catenin / metabolism
Substances
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AR protein, human
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Androgens
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Anilides
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FOXA1 protein, human
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Hepatocyte Nuclear Factor 3-alpha
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Nitriles
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Receptors, Androgen
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Receptors, Estrogen
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Tosyl Compounds
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Wnt Proteins
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beta Catenin
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Dihydrotestosterone
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bicalutamide
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ERBB2 protein, human
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Receptor, ErbB-2