Lymphocytic duodenosis: aetiology and long-term response to specific treatment

Dig Liver Dis. 2012 Aug;44(8):643-8. doi: 10.1016/j.dld.2012.03.006. Epub 2012 Apr 11.

Abstract

Background: The clinical significance of lymphocytic duodenosis remains unclear.

Aim: To prospectively assess the aetiology of lymphocytic duodenosis and the patterns of clinical presentation.

Methods: Ninety consecutive patients with lymphocytic duodenosis and clinical symptoms of the coeliac disease spectrum were prospectively included. All subjects underwent serological testing and HLA genotyping for coeliac disease, assessment of Helicobacter pylori infection, and parasite stool examination. Intake of non-steroidal anti-inflammatory drugs was also recorded. The final aetiology of lymphocytic duodenosis was evaluated on the basis of the long-term response to specific therapy.

Results: More than one initial potential aetiology was observed in 44% of patients. The final diagnosis was gluten-sensitive enteropathy alone or associated with Helicobacter pylori infection in 43.3%, Helicobacter pylori infection (without gluten-sensitive enteropathy) in 24.4%, non-steroidal anti-inflammatory drugs intake in 5.5%, autoimmune disease in 3.3%, and parasitic infection in 2.2%. Among first degree relatives and patients with chronic diarrhoea, the most common final diagnosis was gluten-sensitive enteropathy. In contrast, in the group presenting with chronic dyspepsia the most common diagnosis was Helicobacter pylori infection ('Diarrhoea' vs 'Dyspepsia' groups, p=0.008).

Conclusions: Lymphocytic duodenosis is often associated with more than one potential initial aetiology. Clinical presentation may be useful to decide the initial therapeutic approach with these patients.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adult
  • Anti-Inflammatory Agents, Non-Steroidal / adverse effects
  • Autoimmune Diseases / complications
  • Blastocystis Infections / complications
  • CD3 Complex / metabolism
  • Celiac Disease / blood
  • Celiac Disease / complications
  • Celiac Disease / drug therapy*
  • Duodenal Diseases / etiology*
  • Duodenal Diseases / immunology
  • Duodenal Diseases / pathology
  • Female
  • GTP-Binding Proteins
  • Genotype
  • HLA-DQ Antigens / genetics
  • Helicobacter Infections / complications*
  • Helicobacter pylori
  • Humans
  • Lymphocyte Count
  • Lymphocytes* / metabolism
  • Male
  • Middle Aged
  • Prospective Studies
  • Protein Glutamine gamma Glutamyltransferase 2
  • Transglutaminases / immunology

Substances

  • Anti-Inflammatory Agents, Non-Steroidal
  • CD3 Complex
  • HLA-DQ Antigens
  • HLA-DQ2 antigen
  • HLA-DQ8 antigen
  • Protein Glutamine gamma Glutamyltransferase 2
  • Transglutaminases
  • GTP-Binding Proteins