Manipulation of cell cycle kinetics: influence on the cytotoxicity of doxorubicin in human breast cancer cells

J Steroid Biochem Mol Biol. 1990 Dec 20;37(6):1097-101. doi: 10.1016/0960-0760(90)90472-w.

Abstract

In vitro exposure of estrogen receptor-negative (ER-) EVSA-T human breast cancer cells to insulin and/or estradiol had no effect on cell cycle distribution, in contrast to a 3-5-fold increase in the percentages of cells in the S-phase of the cell cycle in the ER+ MCF-7 cell line. Estrogen pretreatment of MCF-7 cells followed by incubation with doxorubicin resulted in an augmented inhibition of cell growth compared to unstimulated controls. This delay in growth was accompanied by a decrease in the percentages of cells actively synthesizing DNA, and by an augmented percentage of cells exhibiting a G2M-amount of DNA at the end of a 6-9 day period of culture in complete growth medium.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Breast Neoplasms / drug therapy*
  • Bromodeoxyuridine
  • Cell Cycle* / drug effects
  • Cell Cycle* / genetics
  • DNA / metabolism
  • Doxorubicin / pharmacology*
  • Estradiol / pharmacology
  • Female
  • Flow Cytometry
  • Humans
  • Insulin / pharmacology
  • Kinetics
  • Receptors, Estrogen / drug effects
  • Tumor Cells, Cultured

Substances

  • Insulin
  • Receptors, Estrogen
  • Estradiol
  • Doxorubicin
  • DNA
  • Bromodeoxyuridine