Mutations in the ADAR1 gene in Chinese families with dyschromatosis symmetrica hereditaria

Genet Mol Res. 2013 Jan 4;12(3):2794-9. doi: 10.4238/2013.January.4.18.

Abstract

We investigated 2 Chinese families with dyschromatosis symmetrica hereditaria (DSH) and search for mutations in the adenosine deaminase acting on RNA1 (ADAR1) gene in these 2 pedigrees. We performed a mutation analysis of the ADAR1 gene in 2 Chinese families with DSH and reviewed all articles published regarding ADAR1 mutations reported since 2003 by using PubMed. By direct sequencing, a 2-nucleotide AG deletion, 2099-2100delAG, was found in family 1, and a C→T mutation was identified at nucleotide 1420 that changed codon 474 from arginine to a translational termination codon in family 2. Two different pathogenic mutations were identified, c.2099-2100delAG and c.1420C>T, the former being a novel mutation, and the latter previously reported in 3 other families with DSH. To date, a total of 110 mutations in the ADAR1 gene have been reported, and 10 of them were recurrent; the mutations R474X, R1083C, R1096X, and R1155W might be the DSH-related hotspots.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adenosine Deaminase / genetics*
  • Adolescent
  • Adult
  • Child
  • China
  • Female
  • Genes, Dominant
  • Humans
  • Male
  • Mutation*
  • Pedigree
  • Pigmentation Disorders / congenital*
  • Pigmentation Disorders / diagnosis
  • Pigmentation Disorders / genetics
  • RNA-Binding Proteins

Substances

  • RNA-Binding Proteins
  • ADARB1 protein, human
  • Adenosine Deaminase

Supplementary concepts

  • Dyschromatosis symmetrica hereditaria 1