The thymic medulla is required for Foxp3+ regulatory but not conventional CD4+ thymocyte development

J Exp Med. 2013 Apr 8;210(4):675-81. doi: 10.1084/jem.20122070. Epub 2013 Mar 25.

Abstract

A key role of the thymic medulla is to negatively select autoreactive CD4(+) and CD8(+) thymocytes, a process important for T cell tolerance induction. However, the involvement of the thymic medulla in other aspects of αβ T cell development, including the generation of Foxp3(+) natural regulatory T cells (nTreg cells) and the continued maturation of positively selected conventional αβ T cells, is unclear. We show that newly generated conventional CD69(+)Qa2(-) CD4 single-positive thymocytes mature to the late CD69(-)Qa2(+) stage in the absence of RelB-dependent medullary thymic epithelial cells (mTECs). Furthermore, an increasing ability to continue maturation extrathymically is observed within the CD69(+)CCR7(-/lo)CCR9(+) subset of conventional SP4 thymocytes, providing evidence for an independence from medullary support by the earliest stages after positive selection. In contrast, Foxp3(+) nTreg cell development is medullary dependent, with mTECs fostering the generation of Foxp3(-)CD25(+) nTreg cell precursors at the CD69(+)CCR7(+)CCR9(-) stage. Our results demonstrate a differential requirement for the thymic medulla in relation to CD4 conventional and Foxp3(+) thymocyte lineages, in which an intact mTEC compartment is a prerequisite for Foxp3(+) nTreg cell development through the generation of Foxp3(-)CD25(+) nTreg cell precursors.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Antigens, CD / genetics
  • Antigens, CD / immunology
  • Antigens, Differentiation, T-Lymphocyte / genetics
  • Antigens, Differentiation, T-Lymphocyte / immunology
  • Cell Differentiation / physiology*
  • Forkhead Transcription Factors / genetics
  • Forkhead Transcription Factors / immunology*
  • Histocompatibility Antigens Class I / genetics
  • Histocompatibility Antigens Class I / immunology
  • Lectins, C-Type / genetics
  • Lectins, C-Type / immunology
  • Mice
  • Mice, Knockout
  • Receptors, Antigen, T-Cell, alpha-beta / genetics
  • Receptors, Antigen, T-Cell, alpha-beta / immunology
  • Receptors, CCR / genetics
  • Receptors, CCR / immunology
  • Receptors, CCR7 / genetics
  • Receptors, CCR7 / immunology
  • T-Lymphocytes, Regulatory / cytology
  • T-Lymphocytes, Regulatory / immunology*
  • Thymocytes / cytology
  • Thymocytes / immunology*
  • Thymus Gland / cytology
  • Thymus Gland / immunology*
  • Transcription Factor RelB / genetics
  • Transcription Factor RelB / immunology

Substances

  • Antigens, CD
  • Antigens, Differentiation, T-Lymphocyte
  • CC chemokine receptor 9
  • CD69 antigen
  • Ccr7 protein, mouse
  • Forkhead Transcription Factors
  • Foxp3 protein, mouse
  • Histocompatibility Antigens Class I
  • Lectins, C-Type
  • Q surface antigens
  • Receptors, Antigen, T-Cell, alpha-beta
  • Receptors, CCR
  • Receptors, CCR7
  • Relb protein, mouse
  • Transcription Factor RelB