Anti-aging effects of co-enzyme Q10 on periodontal tissues

J Dent Res. 2013 Aug;92(8):735-9. doi: 10.1177/0022034513490959. Epub 2013 May 21.

Abstract

Oxidative stress is associated with age-related reactions. The anti-oxidative effects of a reduced form of co-enzyme Q10 (rCoQ10) suppress oxidative stress, which may contribute to the prevention of age-related inflammatory reactions. We examined the effects of topically applied rCoQ10 on periodontal inflammatory reactions in a rat aging model. Male Fischer 344 rats, 2 (n = 6) and 4 mos (n = 18) of age, were used. All of the two-month-old rats and 6 of the four-month-old rats were sacrificed and 12 remaining four-month-old rats received topically applied ointment with or without 1% rCoQ10 on the gingival surface until they reached 6 mos of age. The rats showed an age-dependent increase in circulating oxidative stress. RCoQ10 decreased oxidative DNA damage and tartrate-resistant acid-phosphatase-positive osteoclasts in the periodontal tissue at 6 mos of age as compared with the control. The same conditions lowered gene expression of caspase-1 and interleukin-1β in the periodontal tissue. Furthermore, Nod-like receptor protein 3 inflammasomes were less activated in periodontal tissues from rCoQ10-treated rats as compared with the control rats. Our results suggest that rCoQ10 suppresses age-related inflammatory reactions and osteoclast differentiation by inhibiting oxidative stress.

Keywords: aging; coenzyme Q10; inflammasomes; oxidative stress; periodontium; preclinical drug evaluation.

Publication types

  • Comparative Study
  • Research Support, Non-U.S. Gov't

MeSH terms

  • 8-Hydroxy-2'-Deoxyguanosine
  • Acid Phosphatase / analysis
  • Actins / drug effects
  • Age Factors
  • Aging / drug effects*
  • Alveolar Process / drug effects
  • Alveolar Process / pathology
  • Animals
  • Antioxidants / pharmacology
  • Apoptosis Regulatory Proteins
  • CARD Signaling Adaptor Proteins
  • Carrier Proteins
  • Caspase 1 / drug effects
  • Cytoskeletal Proteins / drug effects
  • DNA Damage / drug effects
  • Deoxyguanosine / analogs & derivatives
  • Deoxyguanosine / analysis
  • Deoxyguanosine / blood
  • Electron Transport Chain Complex Proteins / pharmacology*
  • Gingiva / drug effects
  • Gingiva / pathology
  • Inflammasomes / drug effects
  • Interleukin-1beta / drug effects
  • Isoenzymes / analysis
  • Male
  • Models, Animal
  • NF-kappa B / drug effects
  • NLR Family, Pyrin Domain-Containing 3 Protein
  • Osteoclasts / drug effects
  • Oxidative Stress / drug effects
  • Periodontium / drug effects*
  • Periodontium / pathology
  • Random Allocation
  • Rats
  • Rats, Inbred F344
  • Receptors, Cytoplasmic and Nuclear / drug effects
  • Tartrate-Resistant Acid Phosphatase
  • Ubiquinone / analogs & derivatives*
  • Ubiquinone / blood
  • Ubiquinone / pharmacology
  • Vitamins / pharmacology*

Substances

  • Actins
  • Antioxidants
  • Apoptosis Regulatory Proteins
  • CARD Signaling Adaptor Proteins
  • Carrier Proteins
  • Cytoskeletal Proteins
  • Electron Transport Chain Complex Proteins
  • Inflammasomes
  • Interleukin-1beta
  • Isoenzymes
  • NF-kappa B
  • NLR Family, Pyrin Domain-Containing 3 Protein
  • Nlrp3 protein, rat
  • Pycard protein, rat
  • Receptors, Cytoplasmic and Nuclear
  • Vitamins
  • Ubiquinone
  • 8-Hydroxy-2'-Deoxyguanosine
  • Acid Phosphatase
  • Tartrate-Resistant Acid Phosphatase
  • Caspase 1
  • coenzyme Q10
  • Deoxyguanosine