Vitamin E isoforms as modulators of lung inflammation

Nutrients. 2013 Oct 31;5(11):4347-63. doi: 10.3390/nu5114347.

Abstract

Asthma and allergic diseases are complex conditions caused by a combination of genetic and environmental factors. Clinical studies suggest a number of protective dietary factors for asthma, including vitamin E. However, studies of vitamin E in allergy commonly result in seemingly conflicting outcomes. Recent work indicates that allergic inflammation is inhibited by supplementation with the purified natural vitamin E isoform α-tocopherol but elevated by the isoform γ-tocopherol when administered at physiological tissue concentrations. In this review, we discuss opposing regulatory effects of α-tocopherol and γ-tocopherol on allergic lung inflammation in clinical trials and in animal studies. A better understanding of the differential regulation of inflammation by isoforms of vitamin E provides a basis towards the design of clinical studies and diets that would effectively modulate inflammatory pathways in lung disease.

Publication types

  • Research Support, N.I.H., Extramural
  • Research Support, Non-U.S. Gov't
  • Review

MeSH terms

  • Animals
  • Antioxidants / adverse effects
  • Antioxidants / pharmacology
  • Antioxidants / therapeutic use
  • Asthma* / etiology
  • Asthma* / prevention & control
  • Diet
  • Dietary Supplements
  • Humans
  • Immunologic Factors / adverse effects
  • Immunologic Factors / pharmacology*
  • Immunologic Factors / therapeutic use
  • Inflammation* / etiology
  • Inflammation* / prevention & control
  • Lung / drug effects*
  • Pneumonia* / etiology
  • Pneumonia* / prevention & control
  • Protein Isoforms
  • alpha-Tocopherol / pharmacology*
  • alpha-Tocopherol / therapeutic use
  • gamma-Tocopherol / adverse effects
  • gamma-Tocopherol / pharmacology*

Substances

  • Antioxidants
  • Immunologic Factors
  • Protein Isoforms
  • gamma-Tocopherol
  • alpha-Tocopherol