L-asparaginase induces in AML U937 cells apoptosis via an AIF-mediated mechanism

Front Biosci (Landmark Ed). 2014 Jan 1;19(3):515-27. doi: 10.2741/4222.

Abstract

Acute Myeloid Leukemia (AML), a cancer of the myeloid line of blood cells, progresses rapidly and is typically fatal within weeks or months if left untreated. Asparaginases are a class of enzymatic anti-leukemia agents that induce apoptosis in leukemia cell lines; however, the role of L-asparaginase in the induction of apoptosis in AML cells has not been investigated. In this study, we investigated the apoptosis-inducing effect of L-asparaginase and its underlying mechanism in AML U937 cells. The results showed that L-asparaginase significantly inhibited the proliferation of U937 cells by inducing apoptosis. Furthermore, the low baseline expression level of asparaginase synthase (ASNS) demonstrated the sensitivity of U937 cells and AML M5, a rare subtype of AML, to L-asparaginase. Apoptosis induced by L-asparaginase is mediated by apoptosis-inducing factor (AIF). Our findings show the potential of L-asparaginase as an effective approach in treating AML via the induction of apoptosis mediated by AIF.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Antineoplastic Agents / pharmacology*
  • Apoptosis / drug effects*
  • Apoptosis Inducing Factor / physiology*
  • Asparaginase / pharmacology*
  • Base Sequence
  • DNA Primers
  • Electrophoresis, Agar Gel
  • Flow Cytometry
  • Humans
  • Leukemia, Myeloid, Acute / drug therapy*
  • Leukemia, Myeloid, Acute / pathology
  • Mice
  • Mice, Inbred NOD
  • Mice, SCID
  • Polymerase Chain Reaction
  • Survival Rate
  • U937 Cells

Substances

  • Antineoplastic Agents
  • Apoptosis Inducing Factor
  • DNA Primers
  • Asparaginase