Glucagon-induced changes in fructose 2,6-bisphosphate and 6-phosphofructo-2-kinase in cultured rat foetal hepatocytes

Biochem J. 1989 Feb 1;257(3):795-9. doi: 10.1042/bj2570795.

Abstract

The sensitivity of 6-phosphofructo-2-kinase to glucagon and cyclic AMP was studied during the perinatal period. In liver homogenates from foetal and neonatal rats, incubation with cyclic AMP produced inactivation of 6-phosphofructo-2-kinase 3 h after birth. The maximal effect was obtained 12 h after birth. In primary cultures of hepatocytes from 22-day-old foetuses, glucogon induced an inhibition of 6-phosphofructo-2-kinase that required 45 min to reach the half-maximal effect. Cycloheximide prevented the glucagon-induced changes in this activity from cultured foetal hepatocytes. These results suggest that the adult form of 6-phosphofructo-2-kinase is rapidly induced after birth, probably by the hormonal changes that occur in this period.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Cells, Cultured
  • Cyclic AMP / metabolism
  • Cycloheximide / pharmacology
  • Fructosediphosphates / metabolism*
  • Glucagon / metabolism*
  • Hexosediphosphates / metabolism*
  • Liver / cytology
  • Liver / embryology
  • Liver / metabolism*
  • Phosphofructokinase-2
  • Phosphotransferases / metabolism*
  • Rats
  • Rats, Inbred Strains

Substances

  • Fructosediphosphates
  • Hexosediphosphates
  • fructose 2,6-diphosphate
  • Glucagon
  • Cycloheximide
  • Cyclic AMP
  • Phosphotransferases
  • Phosphofructokinase-2