New targeted therapies such as anti-adhesion molecules, anti-IL-12/23 and anti-Janus kinases are looking toward a more effective treatment of inflammatory bowel disease

Scand J Gastroenterol. 2015 Jan;50(1):113-20. doi: 10.3109/00365521.2014.993700.

Abstract

Antitumor necrosis factor α agents have dramatically changed the management of inflammatory bowel disease (IBD). However, a significant proportion of patients does not respond or lose response over time. Hence, there is an urgent need for new molecules, with different mechanisms of action, and with a targeted and more effective approach. These new drugs include either small molecules or biological agents. We describe the three most promising classes of molecules in the field of IBD: anti-adhesion, anti-interleukin 12/23 and anti-Janus Kinases therapies.

Keywords: Crohn’s disease; adhesion molecules; anti-Janus kinases; anti-integrins; anti-interleukin 12/23; inflammatory bowel disease; ulcerative colitis.

Publication types

  • Review

MeSH terms

  • Antibodies, Monoclonal, Humanized / therapeutic use*
  • Cell Adhesion Molecules / antagonists & inhibitors
  • Humans
  • Immunosuppressive Agents / therapeutic use*
  • Inflammatory Bowel Diseases / drug therapy*
  • Inflammatory Bowel Diseases / immunology
  • Interleukin-12 / antagonists & inhibitors
  • Interleukin-23 / antagonists & inhibitors
  • Janus Kinases / antagonists & inhibitors
  • Molecular Targeted Therapy / methods*
  • Treatment Outcome

Substances

  • Antibodies, Monoclonal, Humanized
  • Cell Adhesion Molecules
  • Immunosuppressive Agents
  • Interleukin-23
  • Interleukin-12
  • Janus Kinases