[Inhibition of S1PR2 activity down-regulates expressions of sphingosine kinase 1 and MCP-1 in rat glomerular mesangial cells under high glucose]

Xi Bao Yu Fen Zi Mian Yi Xue Za Zhi. 2015 Jan;31(1):36-9.
[Article in Chinese]

Abstract

Objective: To investigate the effects of high glucose and the specific antagonist JTE-013 of sphingosine-1-phosphate receptor 2 (S1PR2) on the expressions of sphingosine kinase 1 (Sphk1), S1PR2 and monocyte chemoattractant protein-1 (MCP-1) in rat glomerular mesangial cells.

Methods: The cultured rat GMCs were divided into four groups: normal glucose control group (NG, with 5.5 mmol/L glucose), mannitol group (HM, with 5.5 mmol/L glucose and 24.5 mmol/L mannitol), high glucose group (HG, with 30 mmol/L glucose), JTE-013 group (HJ, with 30 mmol/L glucose and 10 μmol/L JTE-013). The mRNA levels of SphK1, S1PR2 and MCP-1 were determined with real-time quantitative PCR in the cells at 0, 12, 24 and 48 hours, respectively, and the protein expression of MCP-1 in the supernatant was determined with ELISA .

Results: Compared with those in normal glucose, the mRNAs of SphK1 and S1PR2 in rat GMCs under high glucose were down-regulated at 12 hours and were then up-regulated as time went on, and peaked at 48 hours. High glucose significantly enhanced the mRNA expression of MCP-1 at 12 hours, and the expression reached the highest levels at 24 hours, but decreased at 48 hours. The protein expression of MCP-1 in rat GMCs time-dependently increased under high glucose compared with that in NG. After GMCs were treated with 10 μmol/L JTE-013 before exposed to high glucose for 24 hours, the mRNA levels of SphK1, S1PR2 and MCP-1 and the protein expression of MCP-1 significantly decreased compared with those in HG.

Conclusion: Inhibition of S1PR2 activity could down-regulate the expressions of SphK1 and MCP-1 in rat GMCs under high glucose.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Chemokine CCL2 / genetics
  • Chemokine CCL2 / metabolism*
  • Diabetic Nephropathies / enzymology
  • Diabetic Nephropathies / genetics
  • Diabetic Nephropathies / metabolism*
  • Down-Regulation*
  • Glucose / metabolism*
  • Humans
  • Mesangial Cells / enzymology
  • Mesangial Cells / metabolism*
  • Phosphotransferases (Alcohol Group Acceptor) / genetics
  • Phosphotransferases (Alcohol Group Acceptor) / metabolism*
  • Rats
  • Receptors, Lysosphingolipid / genetics
  • Receptors, Lysosphingolipid / metabolism*
  • Sphingosine-1-Phosphate Receptors

Substances

  • Ccl2 protein, rat
  • Chemokine CCL2
  • Receptors, Lysosphingolipid
  • Sphingosine-1-Phosphate Receptors
  • sphingosine-1-phosphate receptor-2, rat
  • Phosphotransferases (Alcohol Group Acceptor)
  • sphingosine kinase
  • Glucose