Impact of Mitochondria-Mediated Apoptosis in U251 Cell Cycle Arrest in G1 Stage and Caspase Activation

Med Sci Monit. 2015 Nov 23:21:3629-33. doi: 10.12659/msm.894006.

Abstract

BACKGROUND Most mitochondria-mediated apoptosis has some relevance to the cell cycle, but there is still a lack of investigations about U251 cell cycle in human brain glioma cells. In this study, we aimed to clarify the correlation of mitochondria-mediated apoptosis with the U251 cell cycle and its influence on apoptosis, through observing the impact of mitochondria-mediated apoptosis in U251cell specificity cycle arrest and Caspase activation. MATERIAL AND METHODS AnnexinV/PI and API were used to label the brain glioma cells for flow cytometry analysis of U251 cell apoptosis and cell cycle. RT-PCR and Western blot were performed to detect Caspase-3 and Caspase-9 activation. RESULTS Peripheral blood in stationary phase is not sensitive to apoptosis induction, but U251 cells have obvious apoptosis. Mitochondria-mediated apoptosis mainly occurs in the G1 phase of the cell cycle. Caspase-3 and Caspase-9 mRNAs and proteins expression increased significantly after the cells were treated by mitochondrial apoptosis-related gene Bax induction. CONCLUSIONS Mitochondria-mediated apoptosis is related to the U251 cell cycle with specific G1 stage arrest. Caspase activation occurs in the process of cell apoptosis.

MeSH terms

  • Apoptosis / physiology*
  • Brain Neoplasms / enzymology
  • Brain Neoplasms / pathology*
  • Caspase Inhibitors / metabolism*
  • Caspases / metabolism*
  • Cell Line, Tumor
  • Cell Proliferation / physiology
  • G1 Phase Cell Cycle Checkpoints / physiology*
  • Glioma / enzymology
  • Glioma / pathology*
  • Humans
  • Mitochondria / enzymology
  • Mitochondria / physiology*
  • Signal Transduction

Substances

  • Caspase Inhibitors
  • Caspases