Dynamics of Urinary Calprotectin after Renal Ischaemia

PLoS One. 2016 Jan 8;11(1):e0146395. doi: 10.1371/journal.pone.0146395. eCollection 2016.

Abstract

Background: Urinary calprotectin has been identified as a promising biomarker for acute kidney injury. To date, however, the time-dependent changes of this parameter during acute kidney injury remain elusive. The aim of the present work was to define the time-course of urinary calprotectin secretion after ischaemia/reperfusion-induced kidney injury in comparison to neutrophil gelatinase-associated lipocalin, thereby monitoring the extent of tubular damage in nephron sparing surgery for kidney tumours.

Methods: The study population consisted of 42 patients. Thirty-two patients underwent either open or endoscopic nephron sparing surgery for kidney tumours. During the surgery, the renal arterial pedicle was clamped with a median ischaemic time of 13 minutes (interquartile range, 4.5-20.3 minutes) in 26 patients. Ten retro-peritoneoscopic living donor nephrectomy patients and 6 nephron sparing surgery patients in whom the renal artery was not clamped served as controls. Urinary calprotectin and neutrophil gelatinase-associated lipocalin concentrations were repeatedly measured by enzyme-linked immunosorbent assay and assessed according to renal function parameters.

Results: Urinary concentrations of calprotectin and neutrophil gelatinase-associated lipocalin increased significantly after ischaemia/reperfusion injury, whereas concentrations remained unchanged after nephron sparing surgery without ischaemia/reperfusion injury and after kidney donation. Calprotectin and neutrophil gelatinase-associated lipocalin levels were significantly increased 2 and 8 hours, respectively, post-ischaemia. Both proteins reached maximal concentrations after 48 hours, followed by a subsequent persistent decrease. Maximal neutrophil gelatinase-associated lipocalin and calprotectin concentrations were 9-fold and 69-fold higher than their respective baseline values. The glomerular filtration rate was only transiently impaired at the first post-operative day after ischaemia/reperfusion injury (p = 0.049).

Conclusion: Calprotectin and neutrophil gelatinase-associated lipocalin can be used to monitor clinical and sub-clinical tubular damage after nephron sparing surgery for kidney tumours. Urinary calprotectin concentrations start rising within 2 hours after ischaemia/reperfusion-induced kidney injury.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Acute-Phase Proteins / urine*
  • Adult
  • Aged
  • Biomarkers / urine
  • Carcinoma, Renal Cell / blood supply
  • Carcinoma, Renal Cell / pathology
  • Carcinoma, Renal Cell / surgery
  • Carcinoma, Renal Cell / urine*
  • Case-Control Studies
  • Female
  • Glomerular Filtration Rate
  • Humans
  • Kidney / blood supply
  • Kidney / metabolism
  • Kidney / pathology
  • Kidney / surgery
  • Kidney Neoplasms / blood supply
  • Kidney Neoplasms / pathology
  • Kidney Neoplasms / surgery
  • Kidney Neoplasms / urine*
  • Kidney Transplantation*
  • Leukocyte L1 Antigen Complex / urine*
  • Lipocalin-2
  • Lipocalins / urine*
  • Male
  • Middle Aged
  • Neoplasm Staging
  • Nephrectomy / methods
  • Proto-Oncogene Proteins / urine*
  • Renal Artery / surgery
  • Reperfusion Injury / diagnosis
  • Reperfusion Injury / pathology
  • Reperfusion Injury / surgery
  • Reperfusion Injury / urine*
  • Time Factors

Substances

  • Acute-Phase Proteins
  • Biomarkers
  • LCN2 protein, human
  • Leukocyte L1 Antigen Complex
  • Lipocalin-2
  • Lipocalins
  • Proto-Oncogene Proteins

Grants and funding

The study was funded by the German Research Foundation (Research Unit FOR1368, http://www.dfg.de/en/research_funding/). The funders had no role in study design, data collection and analysis, decision to publish, or preparation of the manuscript.