Immunotherapy approaches to treat adult acute lymphoblastic leukemia

Expert Rev Hematol. 2016 Jun;9(6):563-77. doi: 10.1586/17474086.2016.1170593. Epub 2016 Apr 8.

Abstract

Recent developments in immunotherapy are improving treatment results of B-precursor acute lymphoblastic leukemia. This advancement is promoted by new monoclonal antibodies such as inotuzumab ozogamicin, ofatumumab and blinatumomab, by rituximab, and by genetically engineered chimeric antigen receptor-modified T-cells. These treatments, variously targeting CD22, CD20 and CD19 antigens, yield unprecedented high rates of hematologic and molecular remissions even when used in monotherapy and in chemo-resistant or post-transplantation relapsed patients. Beside the encouraging results in relapsed/refractory disease, these agents may open a totally new era in the frontline management of this illness, redefining treatment standards and options for different risk subsets and placing the achievement of a molecular remission at the forefront of treatment objectives. The ever increasing importance of modern immunotherapy in improving treatment design and therapeutic outcome is reviewed.

Keywords: Acute lymphoblastic leukemia; B-cell precursor; blinatumomab; chimeric antigen receptor-modified T cells; hematopoietic cell transplantation; inotuzumab ozogamicin; minimal residual disease; refractory/relapsed; rituximab.

Publication types

  • Review

MeSH terms

  • Adult
  • Animals
  • Antibodies, Monoclonal / pharmacology
  • Antibodies, Monoclonal / therapeutic use
  • Antigens, Neoplasm / genetics
  • Antigens, Neoplasm / metabolism
  • Biomarkers, Tumor / antagonists & inhibitors
  • Combined Modality Therapy
  • Genetic Therapy
  • Humans
  • Immunotherapy* / adverse effects
  • Immunotherapy* / methods
  • Immunotherapy, Adoptive / adverse effects
  • Immunotherapy, Adoptive / methods
  • Molecular Targeted Therapy
  • Neoplasm Staging
  • Precursor Cell Lymphoblastic Leukemia-Lymphoma / diagnosis
  • Precursor Cell Lymphoblastic Leukemia-Lymphoma / therapy*
  • Receptors, Antigen, T-Cell / genetics
  • Receptors, Antigen, T-Cell / metabolism

Substances

  • Antibodies, Monoclonal
  • Antigens, Neoplasm
  • Biomarkers, Tumor
  • Receptors, Antigen, T-Cell